Obesity, metabolic dysfunction, and cardiac fibrosis: pathophysiological pathways, molecular mechanisms, and therapeutic opportunities.

Cavalera, Michele; Wang, Junhong; Frangogiannis, Nikolaos G

Cavalera, Michele; Wang, Junhong; Frangogiannis, Nikolaos G.

Afiliação

Cavalera M; Division of Cardiology, Department of Medicine, The Wilf Cardiovascular Research Institute, Albert Einstein College of Medicine, Bronx, New York.
Wang J; Division of Cardiology, Department of Medicine, The Wilf Cardiovascular Research Institute, Albert Einstein College of Medicine, Bronx, New York.
Frangogiannis NG; Division of Cardiology, Department of Medicine, The Wilf Cardiovascular Research Institute, Albert Einstein College of Medicine, Bronx, New York. Electronic address: ngf@bcm.edu.

Transl Res ; 164(4): 323-35, 2014 Oct.

Article em En | MEDLINE | ID: mdl-24880146

Assuntos

Fibrose/etiologia; Cardiopatias/etiologia; Obesidade/complicações; Animais; Fibrose/patologia; Humanos

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Texto completo: 1 Coleções: 01-internacional Contexto em Saúde: 6_ODS3_enfermedades_notrasmisibles Base de dados: MEDLINE Assunto principal: Fibrose / Cardiopatias / Obesidade Tipo de estudo: Prognostic_studies Limite: Animals / Humans Idioma: En Revista: Transl Res Ano de publicação: 2014 Tipo de documento: Article

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