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LKB1 reduces ROS-mediated cell damage via activation of p38.
Xu, H-G; Zhai, Y-X; Chen, J; Lu, Y; Wang, J-W; Quan, C-S; Zhao, R-X; Xiao, X; He, Q; Werle, K D; Kim, H-G; Lopez, R; Cui, R; Liang, J; Li, Y-L; Xu, Z-X.
Afiliação
  • Xu HG; Division of Hematology and Oncology, Department of Medicine, Comprehensive Cancer Center, University of Alabama at Birmingham, Birmingham, AL, USA.
  • Zhai YX; Key Laboratory of Pathobiology, Ministry of Education, Department of Pathology, Norman Bethune College of Medicine, Jilin University, Changchun, China.
  • Chen J; Division of Hematology and Oncology, Department of Medicine, Comprehensive Cancer Center, University of Alabama at Birmingham, Birmingham, AL, USA.
  • Lu Y; Department of Endocrinology, the Second Affiliated Hospital of Nanjing Medical University, Nanjing, China.
  • Wang JW; Key Laboratory of Pathobiology, Ministry of Education, Department of Pathology, Norman Bethune College of Medicine, Jilin University, Changchun, China.
  • Quan CS; Key Laboratory of Pathobiology, Ministry of Education, Department of Pathology, Norman Bethune College of Medicine, Jilin University, Changchun, China.
  • Zhao RX; Division of Hematology and Oncology, Department of Medicine, Comprehensive Cancer Center, University of Alabama at Birmingham, Birmingham, AL, USA.
  • Xiao X; Division of Hematology and Oncology, Department of Medicine, Comprehensive Cancer Center, University of Alabama at Birmingham, Birmingham, AL, USA.
  • He Q; Division of Hematology and Oncology, Department of Medicine, Comprehensive Cancer Center, University of Alabama at Birmingham, Birmingham, AL, USA.
  • Werle KD; Division of Hematology and Oncology, Department of Medicine, Comprehensive Cancer Center, University of Alabama at Birmingham, Birmingham, AL, USA.
  • Kim HG; Division of Hematology and Oncology, Department of Medicine, Comprehensive Cancer Center, University of Alabama at Birmingham, Birmingham, AL, USA.
  • Lopez R; Division of Hematology and Oncology, Department of Medicine, Comprehensive Cancer Center, University of Alabama at Birmingham, Birmingham, AL, USA.
  • Cui R; Department of Dermatology, Boston University, School of Medicine, Boston, MA, USA.
  • Liang J; Department of Systems Biology, UT MD Anderson Cancer Center, Houston, TX, USA.
  • Li YL; Key Laboratory of Pathobiology, Ministry of Education, Department of Pathology, Norman Bethune College of Medicine, Jilin University, Changchun, China.
  • Xu ZX; 1] Division of Hematology and Oncology, Department of Medicine, Comprehensive Cancer Center, University of Alabama at Birmingham, Birmingham, AL, USA [2] Key Laboratory of Pathobiology, Ministry of Education, Department of Pathology, Norman Bethune College of Medicine, Jilin University, Changchun, C
Oncogene ; 34(29): 3848-59, 2015 Jul.
Article em En | MEDLINE | ID: mdl-25263448
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Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Dano ao DNA / Espécies Reativas de Oxigênio / Proteínas Serina-Treonina Quinases / Proteínas Quinases p38 Ativadas por Mitógeno Limite: Animals / Humans Idioma: En Revista: Oncogene Ano de publicação: 2015 Tipo de documento: Article
Texto completo
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Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Dano ao DNA / Espécies Reativas de Oxigênio / Proteínas Serina-Treonina Quinases / Proteínas Quinases p38 Ativadas por Mitógeno Limite: Animals / Humans Idioma: En Revista: Oncogene Ano de publicação: 2015 Tipo de documento: Article