SIRT1 Protects Against Oxidative Stress-Induced Endothelial Progenitor Cells Apoptosis by Inhibiting FOXO3a via FOXO3a Ubiquitination and Degradation.

Wang, Yu-Qiang; Cao, Qing; Wang, Fei; Huang, Li-Ya; Sang, Tian-Tian; Liu, Fang; Chen, Shu-Yan

Wang, Yu-Qiang; Cao, Qing; Wang, Fei; Huang, Li-Ya; Sang, Tian-Tian; Liu, Fang; Chen, Shu-Yan.

Afiliação

Wang YQ; Department of Geriatrics, Xinhua Hospital Affiliated to Shanghai Jiao Tong University School of Medicine, Shanghai, China.
Cao Q; Department of Geriatrics, Xinhua Hospital Affiliated to Shanghai Jiao Tong University School of Medicine, Shanghai, China.
Wang F; Department of Geriatrics, Xinhua Hospital Affiliated to Shanghai Jiao Tong University School of Medicine, Shanghai, China.
Huang LY; Department of Geriatrics, Xinhua Hospital Affiliated to Shanghai Jiao Tong University School of Medicine, Shanghai, China.
Sang TT; Department of Geriatrics, Xinhua Hospital Affiliated to Shanghai Jiao Tong University School of Medicine, Shanghai, China.
Liu F; Department of Geriatrics, Xinhua Hospital Affiliated to Shanghai Jiao Tong University School of Medicine, Shanghai, China.
Chen SY; Department of Geriatrics, Xinhua Hospital Affiliated to Shanghai Jiao Tong University School of Medicine, Shanghai, China.

J Cell Physiol ; 230(9): 2098-107, 2015 Sep.

Article em En | MEDLINE | ID: mdl-25640014

Assuntos

Apoptose/efeitos dos fármacos; Fatores de Transcrição Forkhead/genética; Estresse Oxidativo/genética; Sirtuína 1/genética; Células Progenitoras Endoteliais/efeitos dos fármacos; Sangue Fetal; Proteína Forkhead Box O3; Fatores de Transcrição Forkhead/antagonistas & inibidores; Humanos; Peróxido de Hidrogênio/administração & dosagem; Proteólise/efeitos dos fármacos; Transdução de Sinais/efeitos dos fármacos; Ativação Transcricional/efeitos dos fármacos; Ubiquitinação/genética

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Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Apoptose / Estresse Oxidativo / Fatores de Transcrição Forkhead / Sirtuína 1 Tipo de estudo: Prognostic_studies Limite: Humans Idioma: En Revista: J Cell Physiol Ano de publicação: 2015 Tipo de documento: Article

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