Deviating the level of proliferating cell nuclear antigen in Trypanosoma brucei elicits distinct mechanisms for inhibiting proliferation and cell cycle progression.
Cell Cycle
; 14(4): 674-88, 2015.
Article
em En
| MEDLINE
| ID: mdl-25701409
Palavras-chave
CDK, cyclin dependent kinase; Cd, Cluster of differentiation; DAPI, 4', 6-diamidino-2-phenylindole; DNA replication; EdU, 5-Ethynyl-2'deoxyuridine; GINS, Go, Ichi, Nii, complex; Gadd, growth arrest and DNA-damage; H2O2, hydrogen peroxide; HU, hydroxyurea; Hs, Homo sapiens; Mcm, mini-chromosome maintenance proteins; MyD, myeloid differentiation primary response gene; Orc, origin recognition complex; PCNA, proliferating cell nuclear antigen; RT-PCR, reverse transcriptase-polymerase chain reaction; Sc, Saccharomyces cerevisiae; Sp, Schizosaccharomyces pombe; Tb, Trypanosoma brucei; attenuate; chemosensitize; hydroxyurea; proliferation
Texto completo:
1
Coleções:
01-internacional
Base de dados:
MEDLINE
Assunto principal:
Trypanosoma brucei brucei
/
Ciclo Celular
/
Antígeno Nuclear de Célula em Proliferação
/
Proliferação de Células
/
Replicação do DNA
/
Pontos de Checagem da Fase G2 do Ciclo Celular
Idioma:
En
Revista:
Cell Cycle
Ano de publicação:
2015
Tipo de documento:
Article