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ON VASCULAR STENOSIS, RESTENOSIS AND MANNOSE BINDING LECTIN.
Kahlow, Barbara Stadler; Nery, Rodrigo Araldi; Skare, Thelma L; Ribas, Carmen Australia Paredes Marcondes; Ramos, Gabriela Piovezani; Petisco, Roberta Dombroski.
Afiliação
  • Kahlow BS; Medical Research Institute, University Evangelic Hospital of Curitiba, Evangelic Faculty of Paraná, Curitiba, PR, Brazil.
  • Nery RA; Medical Research Institute, University Evangelic Hospital of Curitiba, Evangelic Faculty of Paraná, Curitiba, PR, Brazil.
  • Skare TL; Medical Research Institute, University Evangelic Hospital of Curitiba, Evangelic Faculty of Paraná, Curitiba, PR, Brazil.
  • Ribas CA; Medical Research Institute, University Evangelic Hospital of Curitiba, Evangelic Faculty of Paraná, Curitiba, PR, Brazil.
  • Ramos GP; Medical Research Institute, University Evangelic Hospital of Curitiba, Evangelic Faculty of Paraná, Curitiba, PR, Brazil.
  • Petisco RD; Medical Research Institute, University Evangelic Hospital of Curitiba, Evangelic Faculty of Paraná, Curitiba, PR, Brazil.
Arq Bras Cir Dig ; 29(1): 57-9, 2016 Mar.
Article em En, Pt | MEDLINE | ID: mdl-27120743
ABSTRACT
Mannose binding lectin is a lectin instrumental in the innate immunity. It recognizes carbohydrate patterns found on the surface of a large number of pathogenic micro-organisms, activating the complement system. However, this protein seems to increase the tissue damage after ischemia. In this paper is reviewed some aspects of harmful role of the mannose binding lectin in ischemia/reperfusion injury.
Assuntos

Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Traumatismo por Reperfusão / Reestenose Coronária / Lectina de Ligação a Manose Tipo de estudo: Etiology_studies Limite: Humans Idioma: En / Pt Revista: Arq Bras Cir Dig Ano de publicação: 2016 Tipo de documento: Article

Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Traumatismo por Reperfusão / Reestenose Coronária / Lectina de Ligação a Manose Tipo de estudo: Etiology_studies Limite: Humans Idioma: En / Pt Revista: Arq Bras Cir Dig Ano de publicação: 2016 Tipo de documento: Article