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BBC3 in macrophages promoted pulmonary fibrosis development through inducing autophagy during silicosis.
Liu, Haijun; Cheng, Yusi; Yang, Jian; Wang, Wei; Fang, Shencun; Zhang, Wei; Han, Bing; Zhou, Zewei; Yao, Honghong; Chao, Jie; Liao, Hong.
Afiliação
  • Liu H; Neurobiology Laboratory, New Drug Screening Centre, China Pharmaceutical University, Nanjing, China.
  • Cheng Y; Department of Physiology, School of Medicine, Southeast University, Nanjing, China.
  • Yang J; Department of Physiology, School of Medicine, Southeast University, Nanjing, China.
  • Wang W; Department of Physiology, School of Medicine, Southeast University, Nanjing, China.
  • Fang S; Nine Department of Respiratory Medicine, Nanjing Chest Hospital, Nanjing, China.
  • Zhang W; Nine Department of Respiratory Medicine, Nanjing Chest Hospital, Nanjing, China.
  • Han B; Department of Physiology, School of Medicine, Southeast University, Nanjing, China.
  • Zhou Z; Department of Pharmacology, School of Medicine, Southeast University, Nanjing, China.
  • Yao H; Department of Physiology, School of Medicine, Southeast University, Nanjing, China.
  • Chao J; Department of Pharmacology, School of Medicine, Southeast University, Nanjing, China.
  • Liao H; Department of Pharmacology, School of Medicine, Southeast University, Nanjing, China.
Cell Death Dis ; 8(3): e2657, 2017 03 09.
Article em En | MEDLINE | ID: mdl-28277537

Texto completo: 1 Coleções: 01-internacional Contexto em Saúde: 6_ODS3_enfermedades_notrasmisibles Base de dados: MEDLINE Assunto principal: Fibrose Pulmonar / Silicose / Autofagia / Proteínas Proto-Oncogênicas / Proteínas Reguladoras de Apoptose Tipo de estudo: Prognostic_studies Limite: Animals / Humans Idioma: En Revista: Cell Death Dis Ano de publicação: 2017 Tipo de documento: Article

Texto completo: 1 Coleções: 01-internacional Contexto em Saúde: 6_ODS3_enfermedades_notrasmisibles Base de dados: MEDLINE Assunto principal: Fibrose Pulmonar / Silicose / Autofagia / Proteínas Proto-Oncogênicas / Proteínas Reguladoras de Apoptose Tipo de estudo: Prognostic_studies Limite: Animals / Humans Idioma: En Revista: Cell Death Dis Ano de publicação: 2017 Tipo de documento: Article