Novel PRSS1 Mutation p.P17T Validates Pathogenic Relevance of CTRC-Mediated Processing of the Trypsinogen Activation Peptide in Chronic Pancreatitis.

Németh, Balázs Csaba; Szücs, Ákos; Hegyi, Péter; Sahin-Tóth, Miklós

Németh, Balázs Csaba; Szücs, Ákos; Hegyi, Péter; Sahin-Tóth, Miklós.

Afiliação

Németh BC; Center for Exocrine Disorders, Department of Molecular and Cell Biology, Boston University Henry M. Goldman School of Dental Medicine, Boston, Massachusetts, USA.
Szücs Á; First Department of Medicine, University of Szeged, Szeged, Hungary.
Hegyi P; First Department of Surgery, Semmelweis University, Budapest, Hungary.
Sahin-Tóth M; Institute for Translational Medicine and First Department of Medicine, University of Pécs, Pécs, Hungary.

Am J Gastroenterol ; 112(12): 1896-1898, 2017 12.

Article em En | MEDLINE | ID: mdl-29215622

Assuntos

Quimotripsina/metabolismo; Pancreatite/genética; Tripsina/genética; Tripsinogênio/metabolismo; Adulto; Feminino; Humanos; Mutação

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Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Pancreatite / Tripsinogênio / Quimotripsina / Tripsina Limite: Adult / Female / Humans Idioma: En Revista: Am J Gastroenterol Ano de publicação: 2017 Tipo de documento: Article

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