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ß3-adrenergic receptor activation induces TGFß1 expression in cardiomyocytes via the PKG/JNK/c-Jun pathway.
Xu, Zhongcheng; Wu, Jimin; Xin, Junzhou; Feng, Yenan; Hu, Guomin; Shen, Jing; Li, Mingzhe; Zhang, Youyi; Xiao, Han; Wang, Li.
Afiliação
  • Xu Z; The 3rd Department of Cardiology, The First Affiliated Hospital of the Medical College, Shihezi University, Shihezi City, Xinjiang, 832008, China; Department of Cardiology and Institute of Vascular Medicine, Peking University Third Hospital, Key Laboratory of Cardiovascular Molecular Biology and Reg
  • Wu J; Department of Cardiology and Institute of Vascular Medicine, Peking University Third Hospital, Key Laboratory of Cardiovascular Molecular Biology and Regulatory Peptides, Ministry of Health, Key Laboratory of Molecular Cardiovascular Science, Ministry of Education, Beijing Key Laboratory of Cardiova
  • Xin J; The 3rd Department of Cardiology, The First Affiliated Hospital of the Medical College, Shihezi University, Shihezi City, Xinjiang, 832008, China; Department of Cardiology and Institute of Vascular Medicine, Peking University Third Hospital, Key Laboratory of Cardiovascular Molecular Biology and Reg
  • Feng Y; Department of Cardiology and Institute of Vascular Medicine, Peking University Third Hospital, Key Laboratory of Cardiovascular Molecular Biology and Regulatory Peptides, Ministry of Health, Key Laboratory of Molecular Cardiovascular Science, Ministry of Education, Beijing Key Laboratory of Cardiova
  • Hu G; Department of Cardiology and Institute of Vascular Medicine, Peking University Third Hospital, Key Laboratory of Cardiovascular Molecular Biology and Regulatory Peptides, Ministry of Health, Key Laboratory of Molecular Cardiovascular Science, Ministry of Education, Beijing Key Laboratory of Cardiova
  • Shen J; Department of Cardiology and Institute of Vascular Medicine, Peking University Third Hospital, Key Laboratory of Cardiovascular Molecular Biology and Regulatory Peptides, Ministry of Health, Key Laboratory of Molecular Cardiovascular Science, Ministry of Education, Beijing Key Laboratory of Cardiova
  • Li M; Department of Cardiology and Institute of Vascular Medicine, Peking University Third Hospital, Key Laboratory of Cardiovascular Molecular Biology and Regulatory Peptides, Ministry of Health, Key Laboratory of Molecular Cardiovascular Science, Ministry of Education, Beijing Key Laboratory of Cardiova
  • Zhang Y; Department of Cardiology and Institute of Vascular Medicine, Peking University Third Hospital, Key Laboratory of Cardiovascular Molecular Biology and Regulatory Peptides, Ministry of Health, Key Laboratory of Molecular Cardiovascular Science, Ministry of Education, Beijing Key Laboratory of Cardiova
  • Xiao H; Department of Cardiology and Institute of Vascular Medicine, Peking University Third Hospital, Key Laboratory of Cardiovascular Molecular Biology and Regulatory Peptides, Ministry of Health, Key Laboratory of Molecular Cardiovascular Science, Ministry of Education, Beijing Key Laboratory of Cardiova
  • Wang L; The 3rd Department of Cardiology, The First Affiliated Hospital of the Medical College, Shihezi University, Shihezi City, Xinjiang, 832008, China. Electronic address: 183304153@qq.com.
Biochem Biophys Res Commun ; 503(1): 146-151, 2018 09 03.
Article em En | MEDLINE | ID: mdl-29859189
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Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Proteínas Quinases Dependentes de GMP Cíclico / Receptores Adrenérgicos beta 3 / Miócitos Cardíacos / Proteínas Quinases JNK Ativadas por Mitógeno / Fator de Crescimento Transformador beta1 Tipo de estudo: Etiology_studies Limite: Animals Idioma: En Revista: Biochem Biophys Res Commun Ano de publicação: 2018 Tipo de documento: Article
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Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Proteínas Quinases Dependentes de GMP Cíclico / Receptores Adrenérgicos beta 3 / Miócitos Cardíacos / Proteínas Quinases JNK Ativadas por Mitógeno / Fator de Crescimento Transformador beta1 Tipo de estudo: Etiology_studies Limite: Animals Idioma: En Revista: Biochem Biophys Res Commun Ano de publicação: 2018 Tipo de documento: Article