EPHA3 enhances macrophage autophagy and apoptosis by disrupting the mTOR signaling pathway in mice with endometriosis.

Xu, Hongmei; Gao, Yongmei; Shu, Yang; Wang, Yi; Shi, Qingyang

Xu, Hongmei; Gao, Yongmei; Shu, Yang; Wang, Yi; Shi, Qingyang.

Afiliação

Xu H; Department of Obstetrics, The First Hospital of Jilin University, Changchun 130021, Jilin, China.
Gao Y; Department of Obstetrics, The First Hospital of Jilin University, Changchun 130021, Jilin, China.
Shu Y; Department of Gynaecology and Obstetrics, The First Hospital of Jilin University, Changchun 130021, Jilin, China.
Wang Y; Department of Gynaecology and Obstetrics, The First Hospital of Jilin University, Changchun 130021, Jilin, China.
Shi Q; Centre for Reproductive Medicine, Centre for Prenatal Diagnosis, The First Hospital of Jilin University, Changchun 130021, Jilin, China shiqingyang1111@163.com.

Biosci Rep ; 39(7)2019 07 31.

Article em En | MEDLINE | ID: mdl-31262977

Assuntos

Apoptose; Morte Celular Autofágica; Endometriose/metabolismo; Macrófagos/metabolismo; Receptor EphA3/metabolismo; Transdução de Sinais; Serina-Treonina Quinases TOR/metabolismo; Animais; Modelos Animais de Doenças; Endometriose/genética; Endometriose/patologia; Feminino; Macrófagos/patologia; Camundongos; Camundongos Endogâmicos BALB C; Receptor EphA3/genética; Serina-Treonina Quinases TOR/genética

Palavras-chave

Apoptosis; Autophagy; EPHA3; Endometriosis; Macrophage; mTOR signaling pathway

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Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Transdução de Sinais / Apoptose / Receptor EphA3 / Endometriose / Serina-Treonina Quinases TOR / Morte Celular Autofágica / Macrófagos Tipo de estudo: Prognostic_studies Limite: Animals Idioma: En Revista: Biosci Rep Ano de publicação: 2019 Tipo de documento: Article

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