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The Upregulation of Toll-Like Receptor 3 via Autocrine IFN-ß Signaling Drives the Senescence of Human Umbilical Cord Blood-Derived Mesenchymal Stem Cells Through JAK1.
Lee, Hyang Ju; Choi, Bongkun; Kim, Yongsub; Lee, Sang Eun; Jin, Hye Jin; Lee, Hee-Seop; Chang, Eun-Ju; Kim, Seong Who.
Afiliação
  • Lee HJ; Department of Biochemistry and Molecular Biology, Asan Medical Center, University of Ulsan College of Medicine, Seoul, South Korea.
  • Choi B; Stem Cell Immunomodulation Research Center, Asan Medical Center, University of Ulsan College of Medicine, Seoul, South Korea.
  • Kim Y; Stem Cell Immunomodulation Research Center, Asan Medical Center, University of Ulsan College of Medicine, Seoul, South Korea.
  • Lee SE; Department of Biomedical Sciences, Asan Medical Center, University of Ulsan College of Medicine, Seoul, South Korea.
  • Jin HJ; Stem Cell Immunomodulation Research Center, Asan Medical Center, University of Ulsan College of Medicine, Seoul, South Korea.
  • Lee HS; Department of Biomedical Sciences, Asan Medical Center, University of Ulsan College of Medicine, Seoul, South Korea.
  • Chang EJ; Department of Biochemistry and Molecular Biology, Asan Medical Center, University of Ulsan College of Medicine, Seoul, South Korea.
  • Kim SW; Stem Cell Immunomodulation Research Center, Asan Medical Center, University of Ulsan College of Medicine, Seoul, South Korea.
Front Immunol ; 10: 1659, 2019.
Article em En | MEDLINE | ID: mdl-31396213
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Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Interleucina-6 / Senescência Celular / Comunicação Autócrina / Receptor 3 Toll-Like / Janus Quinase 1 / Células-Tronco Mesenquimais Tipo de estudo: Prognostic_studies Limite: Humans Idioma: En Revista: Front Immunol Ano de publicação: 2019 Tipo de documento: Article
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Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Interleucina-6 / Senescência Celular / Comunicação Autócrina / Receptor 3 Toll-Like / Janus Quinase 1 / Células-Tronco Mesenquimais Tipo de estudo: Prognostic_studies Limite: Humans Idioma: En Revista: Front Immunol Ano de publicação: 2019 Tipo de documento: Article