Mito-TIPTP Increases Mitochondrial Function by Repressing the Rubicon-p22phox Interaction in Colitis-Induced Mice.

Kim, Jae-Sung; Kim, Ye-Ram; Jang, Sein; Wang, Sang Geon; Cho, Euni; Mun, Seok-Jun; Jeon, Hye-In; Kim, Hyo-Keun; Min, Sun-Joon; Yang, Chul-Su

Kim, Jae-Sung; Kim, Ye-Ram; Jang, Sein; Wang, Sang Geon; Cho, Euni; Mun, Seok-Jun; Jeon, Hye-In; Kim, Hyo-Keun; Min, Sun-Joon; Yang, Chul-Su.

Afiliação

Kim JS; Department of Bionano Technology, Hanyang University, Seoul 04673, Korea.
Kim YR; Institute of Natural Science & Technology, Hanyang University, Ansan 15588, Korea.
Jang S; Department of Bionano Technology, Hanyang University, Seoul 04673, Korea.
Wang SG; Center for Bionano Intelligence Education and Research, Ansan 15588, Korea.
Cho E; Department of Molecular and Life Science, Hanyang University, Ansan 15588, Korea.
Mun SJ; Center for Bionano Intelligence Education and Research, Ansan 15588, Korea.
Jeon HI; Department of Applied Chemistry, Hanyang University, Ansan 15588, Korea.
Kim HK; Department of Bionano Technology, Hanyang University, Seoul 04673, Korea.
Min SJ; Center for Bionano Intelligence Education and Research, Ansan 15588, Korea.
Yang CS; Department of Bionano Technology, Hanyang University, Seoul 04673, Korea.

Antioxidants (Basel) ; 10(12)2021 Dec 06.

Article em En | MEDLINE | ID: mdl-34943057

Palavras-chave

Rubicon; colitis; mitochondria; p22phox; reactive oxygen species

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Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Tipo de estudo: Prognostic_studies Idioma: En Revista: Antioxidants (Basel) Ano de publicação: 2021 Tipo de documento: Article

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