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SNTA1 gene rescues ion channel function and is antiarrhythmic in cardiomyocytes derived from induced pluripotent stem cells from muscular dystrophy patients.
Jimenez-Vazquez, Eric N; Arad, Michael; Macías, Álvaro; Vera-Pedrosa, Maria L; Cruz, Francisco Miguel; Gutierrez, Lilian K; Cuttitta, Ashley J; Monteiro da Rocha, André; Herron, Todd J; Ponce-Balbuena, Daniela; Guerrero-Serna, Guadalupe; Binah, Ofer; Michele, Daniel E; Jalife, José.
Afiliação
  • Jimenez-Vazquez EN; Department of Internal Medicine and Molecular and Integrative Physiology, Center for Arrhythmia Research, University of Michigan, Ann Arbor, United States.
  • Arad M; Leviev Heart Center, Sheba Medical Center, Tel Hashomer, and Tel Aviv University, Tel Aviv, Israel.
  • Macías Á; Centro Nacional de Investigaciones Cardiovasculares (CNIC) Carlos III, Centro de Investigación Biomédica en Red de Enfermedades Cardiovasculares (CIBERCV), Madrid, Spain.
  • Vera-Pedrosa ML; Centro Nacional de Investigaciones Cardiovasculares (CNIC) Carlos III, Centro de Investigación Biomédica en Red de Enfermedades Cardiovasculares (CIBERCV), Madrid, Spain.
  • Cruz FM; Centro Nacional de Investigaciones Cardiovasculares (CNIC) Carlos III, Centro de Investigación Biomédica en Red de Enfermedades Cardiovasculares (CIBERCV), Madrid, Spain.
  • Gutierrez LK; Centro Nacional de Investigaciones Cardiovasculares (CNIC) Carlos III, Centro de Investigación Biomédica en Red de Enfermedades Cardiovasculares (CIBERCV), Madrid, Spain.
  • Cuttitta AJ; Department of Molecular and Integrative Physiology, University of Michigan Medical School, Ann Arbor, United States.
  • Monteiro da Rocha A; Department of Internal Medicine and Molecular and Integrative Physiology, Center for Arrhythmia Research, University of Michigan, Ann Arbor, United States.
  • Herron TJ; Department of Internal Medicine and Molecular and Integrative Physiology, Center for Arrhythmia Research, University of Michigan, Ann Arbor, United States.
  • Ponce-Balbuena D; Department of Internal Medicine and Molecular and Integrative Physiology, Center for Arrhythmia Research, University of Michigan, Ann Arbor, United States.
  • Guerrero-Serna G; Department of Internal Medicine and Molecular and Integrative Physiology, Center for Arrhythmia Research, University of Michigan, Ann Arbor, United States.
  • Binah O; Department of Physiology, Biophysics and Systems Biology, Ruth and Bruce Rappaport Faculty of Medicine, Technion - Israel Institute of Technology, Haifa, Israel.
  • Michele DE; Department of Molecular and Integrative Physiology, University of Michigan Medical School, Ann Arbor, United States.
  • Jalife J; Department of Internal Medicine and Molecular and Integrative Physiology, Center for Arrhythmia Research, University of Michigan, Ann Arbor, United States.
Elife ; 112022 06 28.
Article em En | MEDLINE | ID: mdl-35762211
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Texto completo: 1 Coleções: 01-internacional Contexto em Saúde: 6_ODS3_enfermedades_notrasmisibles Base de dados: MEDLINE Assunto principal: Proteínas de Ligação ao Cálcio / Distrofia Muscular de Duchenne / Canais de Potássio Corretores do Fluxo de Internalização / Células-Tronco Pluripotentes Induzidas / Proteínas de Membrana / Proteínas Musculares / Cardiomiopatias Limite: Female / Humans / Male Idioma: En Revista: Elife Ano de publicação: 2022 Tipo de documento: Article
Texto completo
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Texto completo: 1 Coleções: 01-internacional Contexto em Saúde: 6_ODS3_enfermedades_notrasmisibles Base de dados: MEDLINE Assunto principal: Proteínas de Ligação ao Cálcio / Distrofia Muscular de Duchenne / Canais de Potássio Corretores do Fluxo de Internalização / Células-Tronco Pluripotentes Induzidas / Proteínas de Membrana / Proteínas Musculares / Cardiomiopatias Limite: Female / Humans / Male Idioma: En Revista: Elife Ano de publicação: 2022 Tipo de documento: Article