Correction: Alzheimer risk gene product Pyk2 suppresses tau phosphorylation and phenotypic effects of tauopathy.

Brody, A Harrison; Nies, Sarah Helena; Guan, Fulin; Smith, Levi M; Mukherjee, Bandhan; Salazar, Santiago A; Lee, Suho; Lam, Tu Kiet T; Strittmatter, Stephen M

Brody, A Harrison; Nies, Sarah Helena; Guan, Fulin; Smith, Levi M; Mukherjee, Bandhan; Salazar, Santiago A; Lee, Suho; Lam, Tu Kiet T; Strittmatter, Stephen M.

Afiliação

Brody AH; Cellular Neuroscience, Neurodegeneration and Repair Program, Departments of Neurology and Neuroscience, Yale School of Medicine, New Haven, CT, USA.
Nies SH; Cellular Neuroscience, Neurodegeneration and Repair Program, Departments of Neurology and Neuroscience, Yale School of Medicine, New Haven, CT, USA.
Guan F; Graduate School of Cellular and Molecular Neuroscience, University of Tübingen, D-72074, Tübingen, Germany.
Smith LM; Cellular Neuroscience, Neurodegeneration and Repair Program, Departments of Neurology and Neuroscience, Yale School of Medicine, New Haven, CT, USA.
Mukherjee B; Cellular Neuroscience, Neurodegeneration and Repair Program, Departments of Neurology and Neuroscience, Yale School of Medicine, New Haven, CT, USA.
Salazar SA; Cellular Neuroscience, Neurodegeneration and Repair Program, Departments of Neurology and Neuroscience, Yale School of Medicine, New Haven, CT, USA.
Lee S; Cellular Neuroscience, Neurodegeneration and Repair Program, Departments of Neurology and Neuroscience, Yale School of Medicine, New Haven, CT, USA.
Lam TKT; Cellular Neuroscience, Neurodegeneration and Repair Program, Departments of Neurology and Neuroscience, Yale School of Medicine, New Haven, CT, USA.
Strittmatter SM; Department of Molecular Biophysics and Biochemistry, Yale School of Medicine, New Haven, CT, USA.

Mol Neurodegener ; 17(1): 56, 2022 Aug 30.

Article em En | MEDLINE | ID: mdl-36038879

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Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Tipo de estudo: Etiology_studies / Risk_factors_studies Idioma: En Revista: Mol Neurodegener Ano de publicação: 2022 Tipo de documento: Article

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