Gene amplification acts as a molecular foothold to facilitate cross-species adaptation and evasion of multiple antiviral pathways.

Banerjee, Shefali; Smith, Cathy; Geballe, Adam P; Rothenburg, Stefan; Kitzman, Jacob O; Brennan, Greg

Banerjee, Shefali; Smith, Cathy; Geballe, Adam P; Rothenburg, Stefan; Kitzman, Jacob O; Brennan, Greg.

Afiliação

Banerjee S; Department of Medical Microbiology and Immunology, School of Medicine, University of California, Davis, CA 95616, USA.
Smith C; Departments of Microbiology and Medicine, University of Washington, Seattle, WA 98195, USA.
Geballe AP; Departments of Human Genetics and Computational Medicine and Bioinformatics, University of Michigan, Ann Arbor, MI 48109, USA.
Rothenburg S; Divisions of Human Biology and Clinical Research, Fred Hutchinson Cancer Center, Seattle, WA 98109, USA.
Kitzman JO; Department of Medical Microbiology and Immunology, School of Medicine, University of California, Davis, CA 95616, USA.
Brennan G; Departments of Microbiology and Medicine, University of Washington, Seattle, WA 98195, USA.

Virus Evol ; 8(2): veac105, 2022.

Article em En | MEDLINE | ID: mdl-36483110

Palavras-chave

PKR; RNase L; RhTRS1; cross-species adaptation; gene duplication; vaccinia virus

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Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Tipo de estudo: Prognostic_studies Idioma: En Revista: Virus Evol Ano de publicação: 2022 Tipo de documento: Article

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