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Acta cir. bras. ; 33(6): 542-550, jun. 2018. tab, graf
Artigo em Inglês | VETINDEX | ID: vti-734723

Resumo

Purpose: To evaluate the effects of 1,25 dihydroxy vitamin D3 (1,25(OH)2D3) on the content of triglyceride (TG), as well as on the gene and protein expressions of adiponectin receptor 2 (AdipoR2), p38 mitogen-activated protein kinase (P38MAPK), and lipoprotein lipase (LPL) in the liver of rats with type 2 diabetes mellitus (T2DM) so as to provide theoretical basis for exploring the mechanism by which 1,25(OH)2D3 regulates TG. Methods: Wistar rats were divided into four groups (n=25), with different treatments and detected the gene and protein expressions of AdipoR2, p38MAPK, and LPL in the liver tissue by reverse transcription polymerase chain reaction (RT-PCR) and Western blotting. Meanwhile, the content of TG in the liver tissue was detected by the Enzyme-linked immunosorbent assay. Results: The expression of AdipoR2, p38MAPK, LPL gene and protein in the liver of VitD intervention group was significantly higher than that in T2DM group (P<0.05), while the TG content was significantly lower than that in T2DM group (P<0.05). Conclusion: 1,25(OH)2D3 can decrease the content of TG in the liver, and its mechanism may be achieved by upregulating the expressions of AdipoR2, p38MAPK, and LPL in the liver.(AU)


Assuntos
Animais , Masculino , Ratos , Calcitriol/uso terapêutico , Triglicerídeos/análise , Regulação da Expressão Gênica , Hepatopatias/terapia , Diabetes Mellitus Tipo 2 , Modelos Animais , Ratos Wistar
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