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1.
Ciênc. rural (Online) ; 52(2): e20200974, 2022. ilus
Artigo em Inglês | VETINDEX, LILACS | ID: biblio-1339656

Resumo

Hypertrophic feline muscular dystrophy (HFMD), rarely reported in the literature, is a disease caused by a hereditary recessive dystrophin deficiency linked to the X chromosome, mainly affecting young male cats. Here, we presented the clinical aspects, food management, and clinical evolution of a seven-year-old mixed-breed cat diagnosed with HFMD, having a primary history of progressive tongue protrusion.


A distrofia muscular hipertrófica felina é uma doença causada por uma deficiência da distrofina com caráter hereditário recessivo ligado ao cromossomo X, com poucos registros de ocorrência na literatura, que acomete principalmente gatos machos jovens. Neste trabalho, são relatados os aspectos clínicos, manejo alimentar e evolução clínica de um gato, sem raça definida, de sete anos com histórico principal de protrusão progressiva da língua e diagnosticado com distrofia muscular hipertrófica felina.


Assuntos
Animais , Masculino , Gatos , Distrofina/genética , Macroglossia/veterinária , Distrofia Muscular Animal/terapia , Biópsia/veterinária
2.
Acta sci. vet. (Impr.) ; 49(suppl.1): Pub.614-Jan 4, 2021. ilus
Artigo em Inglês | VETINDEX | ID: biblio-1458477

Resumo

Background: Extradural synovial cysts (ESC) originate from an extrusion of the synovium in unstable or degeneratedjoints. In the spine, this condition can cause neurological signs such as hyperesthesia, proprioceptive ataxia and paresis.Since extradural presentations of synovial cysts are unusual in dogs, the aim of this manuscript is to report a case ofextradural synovial cyst of the cervical spine, as well as the clinical findings, diagnosis, surgical treatment and clinicalevolution after therapy.Case: A 3-year-old spayed Saint Bernard weighing 60 kg was presented to a Veterinary Medical Teaching Hospital with ahistory of acute paraparesis that evolved to non-ambulatory tetraparesis five days after the appearance of the first clinicalsigns. Neurological examination revealed non-ambulatory tetraparesis, normal muscle tone and segmental spinal reflexesin the thoracic and pelvic limbs, as well as cervical pain associated with limited neck movement. According to the neurological examination, the likely lesion location was the C1-C5 spinal cord segment. The differential diagnosis list includedintervertebral disc disease, caudal cervical spondylomyelopathy, neoplasm, infectious or noninfectious inflammatorydisease, and cystic diseases. Complete blood (cell) count and serum biochemistry tests were within reference limits. Thecerebrospinal fluid analysis revealed 35 mg/dL of protein (< 30 mg/dL) and 27 cells (up to 5 cells/mm3) with a predominance of lymphocytes. In plain radiography, bone proliferations of the C4 (caudal) C5 (cranial) articular processes wereobserved and, in myelography, extradural spinal cord compression was evident between C4-C5 on the right side. The animal underwent dorsal laminectomy for spinal cord decompression. An extradural synovial cyst and proliferated articularprocesses were removed. At 1,281 days after surgery, the dog was clinically normal and presented no neurological deficits...


Assuntos
Feminino , Animais , Cães , Cisto Sinovial/cirurgia , Cisto Sinovial/veterinária , Doenças da Coluna Vertebral/veterinária , Paraparesia/veterinária , Radiculopatia/veterinária , Síndromes de Compressão Nervosa/veterinária , Cães , Laminectomia/veterinária , Vértebras Cervicais
3.
Acta sci. vet. (Online) ; 49(suppl.1): Pub. 614, Mar. 8, 2021. ilus
Artigo em Inglês | VETINDEX | ID: vti-30837

Resumo

Background: Extradural synovial cysts (ESC) originate from an extrusion of the synovium in unstable or degeneratedjoints. In the spine, this condition can cause neurological signs such as hyperesthesia, proprioceptive ataxia and paresis.Since extradural presentations of synovial cysts are unusual in dogs, the aim of this manuscript is to report a case ofextradural synovial cyst of the cervical spine, as well as the clinical findings, diagnosis, surgical treatment and clinicalevolution after therapy.Case: A 3-year-old spayed Saint Bernard weighing 60 kg was presented to a Veterinary Medical Teaching Hospital with ahistory of acute paraparesis that evolved to non-ambulatory tetraparesis five days after the appearance of the first clinicalsigns. Neurological examination revealed non-ambulatory tetraparesis, normal muscle tone and segmental spinal reflexesin the thoracic and pelvic limbs, as well as cervical pain associated with limited neck movement. According to the neurological examination, the likely lesion location was the C1-C5 spinal cord segment. The differential diagnosis list includedintervertebral disc disease, caudal cervical spondylomyelopathy, neoplasm, infectious or noninfectious inflammatorydisease, and cystic diseases. Complete blood (cell) count and serum biochemistry tests were within reference limits. Thecerebrospinal fluid analysis revealed 35 mg/dL of protein (< 30 mg/dL) and 27 cells (up to 5 cells/mm3) with a predominance of lymphocytes. In plain radiography, bone proliferations of the C4 (caudal) C5 (cranial) articular processes wereobserved and, in myelography, extradural spinal cord compression was evident between C4-C5 on the right side. The animal underwent dorsal laminectomy for spinal cord decompression. An extradural synovial cyst and proliferated articularprocesses were removed. At 1,281 days after surgery, the dog was clinically normal and presented no neurological deficits...(AU)


Assuntos
Animais , Feminino , Cães , Cisto Sinovial/cirurgia , Cisto Sinovial/veterinária , Paraparesia/veterinária , Síndromes de Compressão Nervosa/veterinária , Radiculopatia/veterinária , Doenças da Coluna Vertebral/veterinária , Vértebras Cervicais , Cães , Laminectomia/veterinária
4.
Ci. Rural ; 38(3): 830-832, maio-jun. 2008. ilus
Artigo em Inglês | VETINDEX | ID: vti-4373

Resumo

A case of suprasellar germ cell tumor is described in a 6-year -old Airedale Terrier bitch that presented behavioral changes and abrupt onset of blindness. The neoplasm compressed the ventral surface of the cerebrum from the level of basal ganglia to the mesencephalon. Histologically the neoplasm consisted of nests and trabeculae of round to polygonal cells that occasionally surround tubules and cysts formed by columnar cells. Neoplastic cells are immunopositive for cytokeratin and alpha-fetoprotein. The diagnosis was based on location, histological appearance and on the immunohistochemical staining.(AU)


É descrito um caso de tumor de células germinativas supra-selar em numa cadela Airedale Terrier de seis anos de idade, que apresentou transtornos do comportamento e aparecimento abrupto de cegueira. O neoplasma comprimia a superfície ventral do cérebro desde a altura dos núcleos basais até o mesencéfalo. Histologicamente, o neoplasma consistia de ninhos e trabéculas de células redondas ou poligonais que ocasionalmente arranjavam-se ao redor de túbulos e cistos formados por células colunares. As células neoplásicas foram positivas na imunoistoquímica para citoqueratina e a-fetoproteína. O diagnóstico foi feito com base na localização do tumor, no seu aspecto histológico e nos resultados da marcação imunoistoquímica.(AU)


Assuntos
Animais , Cães , Cistos do Sistema Nervoso Central/veterinária , Cães
5.
Ci. Rural ; 38(5): 1375-1380, ago. 2008.
Artigo em Português | VETINDEX | ID: vti-4785

Resumo

O atropelamento por veículos automotivos contribui significativamente para as estatísticas de morte em cães. Entretanto, há poucos estudos sobre os aspectos patológicos reportados na literatura. Este artigo descreve 155 casos fatais de atropelamento por veículos automotivos em cães. Dos 155 cães atropelados, em 138 (89,0 por cento) havia lesões que explicavam a morte ou a razão para a eutanásia desses cães. Essas lesões incluíram traumatismo espinhal-medular (43 [27,7 por cento]), ruptura de órgãos parenquimatosos (40 [25,8 por cento]), traumatismo cranioencefálico (28 [18,1 por cento]), ruptura de órgãos ocos (16 [10,3 por cento]), fratura de costelas com laceração de órgãos parenquimatosos (15 [9,7 por cento]) e ruptura de diafragma com deslocamento de vísceras abdominais para a cavidade torácica (10 [6,4 por cento]).(AU)


Motor vehicle-related trauma significantly contributes to death statistics of dogs. There are however few documented reports on the pathological aspects of such cases. This paper describes 155 fatal cases of dogs victimized by motor vehicle accidents. In 138 (89.0 percent) of the 155 dogs hit by motor vehicles there were lesions that could explain the death or reason for these dogs being euthanatized. These lesions included vertebrospinal trauma (43 [27.7 percent]), rupture of parenchymal organs (40 [25.8 percent]), cranioencephalic trauma (28 [18.1 percent]), rupture of hollow organs (16 [10.3 percent]), rib fracture with subsequent laceration of parenchymal organs (15 [9.7 percent]), and diaphragmatic rupture with displacement of abdominal viscera into the thoracic cavity (10 [6.4 percent]).(AU)


Assuntos
Animais , Cães , Cães , Patologia Veterinária , Traumatologia , Ferimentos e Lesões
6.
MEDVEP. Rev. cient. Med. Vet. ; 6(18): 128-131, abr.-set. 2008. ilus
Artigo em Português | VETINDEX | ID: vti-14608

Resumo

A meningoencefalite necrosante é uma condição inflamatória de cães de raças de pequeno porte especialmente em cães Pug. A doença é caracterizada histologicamente por infiltração de linfócitos, plamócitos e histiócitos e necrose do parênquima cerebral principalmente no córtex. Descreve-se um caso de meningoencefalite necrosante em um cão Pug, fêmea, com cinco anos de idade. O histórico clínico incluía apatia, incoordenação motora e crises epileptiformes recorrentes. O diagnóstico foi realizado com base nas características clínicas, nos achados histopatológicos e na imunoistoquímica(AU)


The necrotizing meningoencephalitis is an inflammatory condition of small dogs of mainly the Pug ones. The disease is histologically characterized by infiltration of lymphocytes, plasma cells and histiocytes and necrosis of the cerebral parenchyma mostly in the cortex. This report concerns a case of necrotizing meningoencephalitis in a female five-year-old Pug dog. The clinical description includes apathy, poorly coordinated gait and recurrent seizures. The diagnosis was carried through on the basis of the clinical, histopathological and immunohistochemical findings(AU)


Assuntos
Cães , Meningoencefalite/veterinária , Cérebro/fisiopatologia , Cães , Necrose , Sistema Nervoso
7.
Artigo em Português | VETINDEX | ID: biblio-1485195

Resumo

A meningoencefalite necrosante é uma condição inflamatória de cães de raças de pequeno porte especialmente em cães Pug. A doença é caracterizada histologicamente por infiltração de linfócitos, plamócitos e histiócitos e necrose do parênquima cerebral principalmente no córtex. Descreve-se um caso de meningoencefalite necrosante em um cão Pug, fêmea, com cinco anos de idade. O histórico clínico incluía apatia, incoordenação motora e crises epileptiformes recorrentes. O diagnóstico foi realizado com base nas características clínicas, nos achados histopatológicos e na imunoistoquímica


The necrotizing meningoencephalitis is an inflammatory condition of small dogs of mainly the Pug ones. The disease is histologically characterized by infiltration of lymphocytes, plasma cells and histiocytes and necrosis of the cerebral parenchyma mostly in the cortex. This report concerns a case of necrotizing meningoencephalitis in a female five-year-old Pug dog. The clinical description includes apathy, poorly coordinated gait and recurrent seizures. The diagnosis was carried through on the basis of the clinical, histopathological and immunohistochemical findings


Assuntos
Cães , Cães , Cérebro/fisiopatologia , Meningoencefalite/veterinária , Necrose , Sistema Nervoso
8.
Tese em Português | VETTESES | ID: vtt-8111

Resumo

Os protocolos de necropsias de cães realizadas no Laboratório de Patologia Veterinária da Universidade Federal de Santa Maria entre 1964 e 2003 foram revisados à procura de lesões hepáticas crônicas. Do total de 4.899 cães necropsiados foram encontradas 306 (6,2%) dessas lesões, que foram classificadas como lesões inflamatórias (4,2%), lesões degenerativas (26,8%), distúrbios circulatórios (23,2%) e tumores (45,8%). Os tumores foram a condição mais comum encontrada nesses cães, e dentre esses, os tumores primários foram os mais freqüentes (43,8%). Colangiocarcinoma foi o tumor primário mais observado (50,0%). Dentre os tumores multicêntricos, o linfoma foi o mais comumente descrito nesses cães (75,0%). Dentre os tumores metastáticos, leucemia e carcinoma mamário foram os mais prevalentes (20,0%). Hiperplasia nodular foi encontrada em 44 (31,4%) cães. Excluindo-se os tumores, a cirrose hepática (26,1%) foi a doença mais importante encontrada nesses cães. Ascite (48,7%) e icterícia (23,7%) foram os sinais clínicos associados à cirrose observados com maior freqüencia. Nos casos em que a enzima alanina aminotransferase foi avaliada, estava elevada em todos os cães com cirrose. Transudato puro foi encontrado em todos os cães com cirrose em que o líquido ascítico foi avaliado. Em 14 cães com cirrose as alterações histológicas foram revistas e a intensidade da fibrose foi dividida em três categorias: fibrose acentuada (grupo 1), fibrose moderada (grupo 2) e fibrose leve (grupo 3). A maioria desses cães (57,2%) apresentou fibrose leve. Outras alterações histológicas observadas nesses casos, em ordem decrescente de freqüência, foram degeneração gordurosa (71,4%), inflamação (71,4%), hemossiderose (64,3%), proliferação de ductos (50,0%), bilestase (42,8%) e necrose de coagulação (35,7%). Fígado com aspecto semelhante à, secundário à insuficiência cardíaca congestiva, foi encontrado em 69 (22,5%) cães. Nos casos em que o líquido abdominal foi avaliado, transudato modificado foi encontrado em todos os cães com insuficiência cardíaca congestiva que apresentaram fígado com aspecto semelhante à noz-moscada


The necropsy reports of 4899 dogs necropsied at the Veterinary Pathology Laboratory of the Universidade Federal de Santa Maria from 1964 to 2003 were reviewed in search of chronic hepatic lesions. Three hundred and six lesions (6,2%) were found and were classified as inflammatory (4,2%), degenerative (26,8%), circulatory (23,2%) and neoplastic (45,8%). Neoplasms were constituted the most common pathology and among them primary tumors were the most prevalent (43,8%). Colangiocarcinoma was the most common primary tumor (50,0%), lymphoma was the most common among multicentric tumors (75,0%) and leukemia and mammary carcinoma were the most prevalent among metastatic tumors (20%). Nodular hyperplasia was found in 44 (31,4%) dogs. Excluding tumors, hepatic cirrhosis was the most important disease of the dogs (26,1%). The clinical signs reported were ascites (48,7%) and icterus (23,7%). In all dogs with cirrhosis when ALT was evaluated, it was elevated. When the ascitic fluid was evaluated in the cirrhotic dogs, it was always pure transudate. In 14 cirrhotic dogs histological changes were reviewed and the intensity of fibrosis was categorized as marked (group 1), moderate (group 2) and mild (group 3). Most of the dogs (57,2%) had mild fibrosis. It was not found a correlation between the extension of fibrosis and the intensity of the clinical signs. Other histological changes in decreasing order of frequency were fatty change (71,4%), inflammation (71,4%), hemosiderosis (64,3%), bile duct proliferation (50,0%), cholestasis (42,8%), and coagulative necrosis (35,7%). Nutmeg liver due to congestive heart failure was found in 69 (22,5%) dogs. When the ascitic fluid of some of the latter dogs was evaluated, it consisted of a modified transudate

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