Detalhe da pesquisa
1.
Parent-of-origin effects implicate epigenetic regulation of experimental autoimmune encephalomyelitis and identify imprinted Dlk1 as a novel risk gene.
PLoS Genet
; 10(3): e1004265, 2014 Mar.
Artigo
em Inglês
| MEDLINE | ID: mdl-24676147
2.
Both MHC and non-MHC genes regulate inflammation and T-cell response after traumatic brain injury.
Brain Behav Immun
; 25(5): 981-90, 2011 Jul.
Artigo
em Inglês
| MEDLINE | ID: mdl-20974248
3.
Advanced intercross line mapping suggests that ncf1 (ean6) regulates severity in an animal model of guillain-barre syndrome.
J Immunol
; 182(7): 4432-8, 2009 Apr 01.
Artigo
em Inglês
| MEDLINE | ID: mdl-19299744
4.
Expression of Ccl11 associates with immune response modulation and protection against neuroinflammation in rats.
PLoS One
; 7(7): e39794, 2012.
Artigo
em Inglês
| MEDLINE | ID: mdl-22815714
5.
Fine-mapping resolves Eae23 into two QTLs and implicates ZEB1 as a candidate gene regulating experimental neuroinflammation in rat.
PLoS One
; 5(9): e12716, 2010 Sep 15.
Artigo
em Inglês
| MEDLINE | ID: mdl-20856809
6.
A role for VAV1 in experimental autoimmune encephalomyelitis and multiple sclerosis.
Sci Transl Med
; 1(10): 10ra21, 2009 Dec 09.
Artigo
em Inglês
| MEDLINE | ID: mdl-20368159