Detalhe da pesquisa
1.
An ERK5-PFKFB3 axis regulates glycolysis and represents a therapeutic vulnerability in pediatric diffuse midline glioma.
Cell Rep
; 43(1): 113557, 2024 01 23.
Artigo
em Inglês
| MEDLINE | ID: mdl-38113141
2.
Machine learning multi-omics analysis reveals cancer driver dysregulation in pan-cancer cell lines compared to primary tumors.
Commun Biol
; 5(1): 1367, 2022 12 13.
Artigo
em Inglês
| MEDLINE | ID: mdl-36513728
3.
Loss of MAT2A compromises methionine metabolism and represents a vulnerability in H3K27M mutant glioma by modulating the epigenome.
Nat Cancer
; 3(5): 629-648, 2022 05.
Artigo
em Inglês
| MEDLINE | ID: mdl-35422502
4.
A Functional Precision Medicine Pipeline Combines Comparative Transcriptomics and Tumor Organoid Modeling to Identify Bespoke Treatment Strategies for Glioblastoma.
Cells
; 10(12)2021 12 02.
Artigo
em Inglês
| MEDLINE | ID: mdl-34943910
5.
Identification of a differentiation stall in epithelial mesenchymal transition in histone H3-mutant diffuse midline glioma.
Gigascience
; 9(12)2020 12 15.
Artigo
em Inglês
| MEDLINE | ID: mdl-33319914
6.
Author Correction: Loss of MAT2A compromises methionine metabolism and represents a vulnerability in H3K27M mutant glioma by modulating the epigenome.
Nat Cancer
; 3(7): 899, 2022 Jul.
Artigo
em Inglês
| MEDLINE | ID: mdl-35739422