Detalhe da pesquisa
1.
T cell immunotherapies engage neutrophils to eliminate tumor antigen escape variants.
Cell
; 186(7): 1432-1447.e17, 2023 03 30.
Artigo
em Inglês
| MEDLINE | ID: mdl-37001503
2.
Clonal Abundance of Tumor-Specific CD4(+) T Cells Potentiates Efficacy and Alters Susceptibility to Exhaustion.
Immunity
; 44(1): 179-193, 2016 Jan 19.
Artigo
em Inglês
| MEDLINE | ID: mdl-26789923
3.
Abnormal mitochondrial transport and morphology are common pathological denominators in SOD1 and TDP43 ALS mouse models.
Hum Mol Genet
; 23(6): 1413-24, 2014 Mar 15.
Artigo
em Inglês
| MEDLINE | ID: mdl-24154542
4.
Anti-tumor activity of a novel LAIR1 antagonist in combination with anti-PD-1 to treat collagen-rich solid tumors.
Mol Cancer Ther
; 2024 Apr 22.
Artigo
em Inglês
| MEDLINE | ID: mdl-38648067
5.
In vivo pathogenic role of mutant SOD1 localized in the mitochondrial intermembrane space.
J Neurosci
; 31(44): 15826-37, 2011 Nov 02.
Artigo
em Inglês
| MEDLINE | ID: mdl-22049426
6.
Sequence of αPD-1 relative to local tumor irradiation determines the induction of abscopal antitumor immune responses.
Sci Immunol
; 6(58)2021 04 09.
Artigo
em Inglês
| MEDLINE | ID: mdl-33837124
7.
Computational algorithm-driven evaluation of monocytic myeloid-derived suppressor cell frequency for prediction of clinical outcomes.
Cancer Immunol Res
; 2(8): 812-21, 2014 Aug.
Artigo
em Inglês
| MEDLINE | ID: mdl-24844912
8.
Monocytic CCR2(+) myeloid-derived suppressor cells promote immune escape by limiting activated CD8 T-cell infiltration into the tumor microenvironment.
Cancer Res
; 72(4): 876-86, 2012 Feb 15.
Artigo
em Inglês
| MEDLINE | ID: mdl-22174368