Detalhe da pesquisa
1.
A proliferative burst during preadolescence establishes the final cardiomyocyte number.
Cell
; 157(4): 795-807, 2014 May 08.
Artigo
em Inglês
| MEDLINE | ID: mdl-24813607
2.
Dysregulation of catalase activity in newborn myocytes during hypoxia is mediated by c-Abl tyrosine kinase.
J Cardiovasc Pharmacol Ther
; 20(1): 93-103, 2015 Jan.
Artigo
em Inglês
| MEDLINE | ID: mdl-24831254
3.
Bioactive nanoparticles improve calcium handling in failing cardiac myocytes.
Nanomedicine (Lond)
; 10(22): 3343-57, 2015 Nov.
Artigo
em Inglês
| MEDLINE | ID: mdl-26223412
4.
Cardioprotection from oxidative stress in the newborn heart by activation of PPARγ is mediated by catalase.
Free Radic Biol Med
; 53(2): 208-15, 2012 Jul 15.
Artigo
em Inglês
| MEDLINE | ID: mdl-22609424
5.
Stretch-activated channel activation promotes early afterdepolarizations in rat ventricular myocytes under oxidative stress.
Am J Physiol Heart Circ Physiol
; 296(5): H1227-35, 2009 May.
Artigo
em Inglês
| MEDLINE | ID: mdl-19286952
6.
Evidence of a genomic biomarker in normal human epithelial mammary cell line, MCF-10A, that is absent in the human breast cancer cell line, MCF-7.
J Biomed Biotechnol
; 2006(2): 43181, 2006.
Artigo
em Inglês
| MEDLINE | ID: mdl-16883051
7.
Dephosphorylation specificities of protein phosphatase for cardiac troponin I, troponin T, and sites within troponin T.
Int J Biol Sci
; 2(1): 1-9, 2006.
Artigo
em Inglês
| MEDLINE | ID: mdl-16585947