Detalhe da pesquisa
1.
GSDMB is increased in IBD and regulates epithelial restitution/repair independent of pyroptosis.
Cell
; 185(2): 283-298.e17, 2022 01 20.
Artigo
em Inglês
| MEDLINE | ID: mdl-35021065
2.
GSDMB is increased in IBD and regulates epithelial restitution/repair independent of pyroptosis.
Cell
; 187(4): 1011-1015, 2024 Feb 15.
Artigo
em Inglês
| MEDLINE | ID: mdl-38364781
3.
Interleukin 33 Triggers Early Eosinophil-Dependent Events Leading to Metaplasia in a Chronic Model of Gastritis-Prone Mice.
Gastroenterology
; 160(1): 302-316.e7, 2021 01.
Artigo
em Inglês
| MEDLINE | ID: mdl-33010253
4.
IL-33 promotes recovery from acute colitis by inducing miR-320 to stimulate epithelial restitution and repair.
Proc Natl Acad Sci U S A
; 115(40): E9362-E9370, 2018 10 02.
Artigo
em Inglês
| MEDLINE | ID: mdl-30224451
5.
A signalling cascade of IL-33 to IL-13 regulates metaplasia in the mouse stomach.
Gut
; 67(5): 805-817, 2018 05.
Artigo
em Inglês
| MEDLINE | ID: mdl-28196875
6.
IL-33 activates tumor stroma to promote intestinal polyposis.
Proc Natl Acad Sci U S A
; 112(19): E2487-96, 2015 May 12.
Artigo
em Inglês
| MEDLINE | ID: mdl-25918379
7.
8.
IL-33 Drives Eosinophil Infiltration and Pathogenic Type 2 Helper T-Cell Immune Responses Leading to Chronic Experimental Ileitis.
Am J Pathol
; 186(4): 885-98, 2016 Apr.
Artigo
em Inglês
| MEDLINE | ID: mdl-26908008
9.
Central role of IL-17/Th17 immune responses and the gut microbiota in the pathogenesis of intestinal fibrosis.
Curr Opin Gastroenterol
; 30(6): 531-8, 2014 Nov.
Artigo
em Inglês
| MEDLINE | ID: mdl-25255234
10.
Mechanisms and models for intestinal fibrosis in IBD.
Dig Dis
; 32 Suppl 1: 26-34, 2014.
Artigo
em Inglês
| MEDLINE | ID: mdl-25531350
11.
The Importance of Th2 Immune Responses in Mediating the Progression of Gastritis-Associated Metaplasia to Gastric Cancer.
Cancers (Basel)
; 16(3)2024 Jan 25.
Artigo
em Inglês
| MEDLINE | ID: mdl-38339273
12.
Candida tropicalis Affects Candida albicans Virulence by Limiting Its Capacity to Adhere to the Host Intestinal Surface, Leading to Decreased Susceptibility to Colitis in Mice.
J Fungi (Basel)
; 10(4)2024 Mar 25.
Artigo
em Inglês
| MEDLINE | ID: mdl-38667916
13.
The role of IL-33 in gut mucosal inflammation.
Mediators Inflamm
; 2013: 608187, 2013.
Artigo
em Inglês
| MEDLINE | ID: mdl-23766561
14.
Candida tropicalis Infection Modulates the Gut Microbiome and Confers Enhanced Susceptibility to Colitis in Mice.
Cell Mol Gastroenterol Hepatol
; 13(3): 901-923, 2022.
Artigo
em Inglês
| MEDLINE | ID: mdl-34890843
15.
NOD2 drives early IL-33-dependent expansion of group 2 innate lymphoid cells during Crohn's disease-like ileitis.
J Clin Invest
; 131(5)2021 03 01.
Artigo
em Inglês
| MEDLINE | ID: mdl-33444291
16.
Cytokine-Mediated Regulation of Innate Lymphoid Cell Plasticity in Gut Mucosal Immunity.
Front Immunol
; 11: 585319, 2020.
Artigo
em Inglês
| MEDLINE | ID: mdl-33424837
17.
Death-Domain-Receptor 3 Deletion Normalizes Inflammatory Gene Expression and Prevents Ileitis in Experimental Crohn's Disease.
Inflamm Bowel Dis
; 25(1): 14-26, 2019 01 01.
Artigo
em Inglês
| MEDLINE | ID: mdl-30295722
18.
The Ugly Duckling of Thiopurines Becomes the Beautiful Swan of Colitis-associated Cancer Management.
Cell Mol Gastroenterol Hepatol
; 11(1): 297-298, 2021.
Artigo
em Inglês
| MEDLINE | ID: mdl-33068529
19.
Central role of the gut epithelial barrier in the pathogenesis of chronic intestinal inflammation: lessons learned from animal models and human genetics.
Front Immunol
; 4: 280, 2013 Sep 17.
Artigo
em Inglês
| MEDLINE | ID: mdl-24062746
20.
Estrogen Receptor α Loss-of-Function Protects Female Mice From DSS-Induced Experimental Colitis.
Cell Mol Gastroenterol Hepatol
; 5(4): 630-633.e1, 2018.
Artigo
em Inglês
| MEDLINE | ID: mdl-29930983