Detalhe da pesquisa
1.
Host-encoded CTCF regulates human cytomegalovirus latency via chromatin looping.
Proc Natl Acad Sci U S A
; 121(10): e2315860121, 2024 Mar 05.
Artigo
em Inglês
| MEDLINE | ID: mdl-38408244
2.
Bromodomain proteins regulate human cytomegalovirus latency and reactivation allowing epigenetic therapeutic intervention.
Proc Natl Acad Sci U S A
; 118(9)2021 03 02.
Artigo
em Inglês
| MEDLINE | ID: mdl-33619107
3.
Short- and long-range cis interactions between integrated HPV genomes and cellular chromatin dysregulate host gene expression in early cervical carcinogenesis.
PLoS Pathog
; 17(8): e1009875, 2021 08.
Artigo
em Inglês
| MEDLINE | ID: mdl-34432858
4.
Killer cell proteases can target viral immediate-early proteins to control human cytomegalovirus infection in a noncytotoxic manner.
PLoS Pathog
; 16(4): e1008426, 2020 04.
Artigo
em Inglês
| MEDLINE | ID: mdl-32282833
5.
Correction: Epstein-Barr virus nuclear antigen EBNA-LP is essential for transforming naïve B cells, and facilitates recruitment of transcription factors to the viral genome.
PLoS Pathog
; 15(2): e1007403, 2019 02.
Artigo
em Inglês
| MEDLINE | ID: mdl-30789958
6.
Disruption of CTCF-YY1-dependent looping of the human papillomavirus genome activates differentiation-induced viral oncogene transcription.
PLoS Biol
; 16(10): e2005752, 2018 10.
Artigo
em Inglês
| MEDLINE | ID: mdl-30359362
7.
Human cytomegalovirus major immediate early transcripts arise predominantly from the canonical major immediate early promoter in reactivating progenitor-derived dendritic cells.
J Gen Virol
; 101(6): 635-644, 2020 06.
Artigo
em Inglês
| MEDLINE | ID: mdl-32375946
8.
Epstein-Barr virus nuclear antigen EBNA-LP is essential for transforming naïve B cells, and facilitates recruitment of transcription factors to the viral genome.
PLoS Pathog
; 14(2): e1006890, 2018 02.
Artigo
em Inglês
| MEDLINE | ID: mdl-29462212
9.
Human papillomavirus genome integration in squamous carcinogenesis: what have next-generation sequencing studies taught us?
J Pathol
; 245(1): 9-18, 2018 05.
Artigo
em Inglês
| MEDLINE | ID: mdl-29443391
10.
Pathogenesis of human papillomavirus-associated mucosal disease.
J Pathol
; 235(4): 527-38, 2015 Mar.
Artigo
em Inglês
| MEDLINE | ID: mdl-25604863
11.
Virus transcript levels and cell growth rates after naturally occurring HPV16 integration events in basal cervical keratinocytes.
J Pathol
; 233(3): 281-93, 2014 Jul.
Artigo
em Inglês
| MEDLINE | ID: mdl-24752734
12.
Depletion of HPV16 early genes induces autophagy and senescence in a cervical carcinogenesis model, regardless of viral physical state.
J Pathol
; 231(3): 354-66, 2013 Nov.
Artigo
em Inglês
| MEDLINE | ID: mdl-23913724
13.
Depletion of polycistronic transcripts using short interfering RNAs: cDNA synthesis method affects levels of non-targeted genes determined by quantitative PCR.
Virol J
; 10: 159, 2013 May 21.
Artigo
em Inglês
| MEDLINE | ID: mdl-23693071
14.
Chromatin control of human cytomegalovirus infection.
mBio
; 14(4): e0032623, 2023 08 31.
Artigo
em Inglês
| MEDLINE | ID: mdl-37439556
15.
Cytomegalovirus US28 regulates cellular EphA2 to maintain viral latency.
Sci Adv
; 8(43): eadd1168, 2022 Oct 28.
Artigo
em Inglês
| MEDLINE | ID: mdl-36288299
16.
A BMPR2/YY1 Signaling Axis Is Required for Human Cytomegalovirus Latency in Undifferentiated Myeloid Cells.
mBio
; 12(3): e0022721, 2021 06 29.
Artigo
em Inglês
| MEDLINE | ID: mdl-34061599
17.
Targeting the latent human cytomegalovirus reservoir for T-cell-mediated killing with virus-specific nanobodies.
Nat Commun
; 12(1): 4436, 2021 07 21.
Artigo
em Inglês
| MEDLINE | ID: mdl-34290252
18.
Latent Cytomegalovirus-Driven Recruitment of Activated CD4+ T Cells Promotes Virus Reactivation.
Front Immunol
; 12: 657945, 2021.
Artigo
em Inglês
| MEDLINE | ID: mdl-33912186
19.
Bromodomain Inhibitors as Therapeutics for Herpesvirus-Related Disease: All BETs Are Off?
Front Cell Infect Microbiol
; 10: 329, 2020.
Artigo
em Inglês
| MEDLINE | ID: mdl-32714883
20.
Lytic infection of permissive cells with human cytomegalovirus is regulated by an intrinsic 'pre-immediate-early' repression of viral gene expression mediated by histone post-translational modification.
J Gen Virol
; 90(Pt 10): 2364-2374, 2009 Oct.
Artigo
em Inglês
| MEDLINE | ID: mdl-19515830