Detalhe da pesquisa
1.
Integrative proteomics highlight presynaptic alterations and c-Jun misactivation as convergent pathomechanisms in ALS.
Acta Neuropathol
; 146(3): 451-475, 2023 09.
Artigo
em Inglês
| MEDLINE | ID: mdl-37488208
2.
Pre-natal manifestation of systemic developmental abnormalities in spinal muscular atrophy.
Hum Mol Genet
; 29(16): 2674-2683, 2020 09 29.
Artigo
em Inglês
| MEDLINE | ID: mdl-32644120
3.
Altered mitochondrial bioenergetics are responsible for the delay in Wallerian degeneration observed in neonatal mice.
Neurobiol Dis
; 130: 104496, 2019 10.
Artigo
em Inglês
| MEDLINE | ID: mdl-31176719
4.
Sideroflexin 3 is an α-synuclein-dependent mitochondrial protein that regulates synaptic morphology.
J Cell Sci
; 130(2): 325-331, 2017 01 15.
Artigo
em Inglês
| MEDLINE | ID: mdl-28049716
5.
Structural basis of PROTAC cooperative recognition for selective protein degradation.
Nat Chem Biol
; 13(5): 514-521, 2017 05.
Artigo
em Inglês
| MEDLINE | ID: mdl-28288108
6.
UBA1/GARS-dependent pathways drive sensory-motor connectivity defects in spinal muscular atrophy.
Brain
; 141(10): 2878-2894, 2018 10 01.
Artigo
em Inglês
| MEDLINE | ID: mdl-30239612
7.
Correction to: Integrative proteomics highlight presynaptic alterations and c-Jun misactivation as convergent pathomechanisms in ALS.
Acta Neuropathol
; 146(5): 783, 2023 Nov.
Artigo
em Inglês
| MEDLINE | ID: mdl-37698628
8.
Proteomic Profiling of Cranial (Superior) Cervical Ganglia Reveals Beta-Amyloid and Ubiquitin Proteasome System Perturbations in an Equine Multiple System Neuropathy.
Mol Cell Proteomics
; 14(11): 3072-86, 2015 Nov.
Artigo
em Inglês
| MEDLINE | ID: mdl-26364976
9.
Protein kinase IKKß-catalyzed phosphorylation of IRF5 at Ser462 induces its dimerization and nuclear translocation in myeloid cells.
Proc Natl Acad Sci U S A
; 111(49): 17432-7, 2014 Dec 09.
Artigo
em Inglês
| MEDLINE | ID: mdl-25326418
10.
Urinary peptidomics in a rodent model of diabetic nephropathy highlights epidermal growth factor as a biomarker for renal deterioration in patients with type 2 diabetes.
Kidney Int
; 89(5): 1125-1135, 2016 May.
Artigo
em Inglês
| MEDLINE | ID: mdl-27083286
11.
Loss of glial neurofascin155 delays developmental synapse elimination at the neuromuscular junction.
J Neurosci
; 34(38): 12904-18, 2014 Sep 17.
Artigo
em Inglês
| MEDLINE | ID: mdl-25232125
12.
Phosphorylation of a splice variant of collapsin response mediator protein 2 in the nucleus of tumour cells links cyclin dependent kinase-5 to oncogenesis.
BMC Cancer
; 15: 885, 2015 Nov 10.
Artigo
em Inglês
| MEDLINE | ID: mdl-26555036
13.
Combining comparative proteomics and molecular genetics uncovers regulators of synaptic and axonal stability and degeneration in vivo.
PLoS Genet
; 8(8): e1002936, 2012.
Artigo
em Inglês
| MEDLINE | ID: mdl-22952455
14.
Label-free quantitative proteomic profiling identifies disruption of ubiquitin homeostasis as a key driver of Schwann cell defects in spinal muscular atrophy.
J Proteome Res
; 13(11): 4546-57, 2014 Nov 07.
Artigo
em Inglês
| MEDLINE | ID: mdl-25151848
15.
Reproducible automated phosphopeptide enrichment using magnetic TiO2 and Ti-IMAC.
Anal Chem
; 86(20): 10296-302, 2014 Oct 21.
Artigo
em Inglês
| MEDLINE | ID: mdl-25233145
16.
Phosphorylation of Arabidopsis UVR8 photoreceptor modulates protein interactions and responses to UV-B radiation.
Nat Commun
; 15(1): 1221, 2024 Feb 09.
Artigo
em Inglês
| MEDLINE | ID: mdl-38336824
17.
Reversible molecular pathology of skeletal muscle in spinal muscular atrophy.
Hum Mol Genet
; 20(22): 4334-44, 2011 Nov 15.
Artigo
em Inglês
| MEDLINE | ID: mdl-21840928
18.
Keap1 perceives stress via three sensors for the endogenous signaling molecules nitric oxide, zinc, and alkenals.
Proc Natl Acad Sci U S A
; 107(44): 18838-43, 2010 Nov 02.
Artigo
em Inglês
| MEDLINE | ID: mdl-20956331
19.
Necroptosis inhibition counteracts neurodegeneration, memory decline, and key hallmarks of aging, promoting brain rejuvenation.
Aging Cell
; 22(5): e13814, 2023 05.
Artigo
em Inglês
| MEDLINE | ID: mdl-36973898
20.
SMN deficiency disrupts brain development in a mouse model of severe spinal muscular atrophy.
Hum Mol Genet
; 19(21): 4216-28, 2010 Nov 01.
Artigo
em Inglês
| MEDLINE | ID: mdl-20705736