Detalhe da pesquisa
1.
Intrinsic bias at non-canonical, ß-arrestin-coupled seven transmembrane receptors.
Mol Cell
; 81(22): 4605-4621.e11, 2021 11 18.
Artigo
em Inglês
| MEDLINE | ID: mdl-34582793
2.
Complement(ing) long-COVID thromboinflammation and pathogenesis.
Trends Immunol
; 2024 Apr 17.
Artigo
em Inglês
| MEDLINE | ID: mdl-38637200
3.
Cell-intrinsic C5a synergizes with Dectin-1 in macrophages to mediate fungal killing.
Proc Natl Acad Sci U S A
; 121(5): e2314627121, 2024 Jan 30.
Artigo
em Inglês
| MEDLINE | ID: mdl-38252818
4.
SARS-CoV-2 drives NLRP3 inflammasome activation in human microglia through spike protein.
Mol Psychiatry
; 28(7): 2878-2893, 2023 Jul.
Artigo
em Inglês
| MEDLINE | ID: mdl-36316366
5.
Unexpected Off-Target Activities for Recombinant C5a in Human Macrophages.
J Immunol
; 208(1): 133-142, 2022 01 01.
Artigo
em Inglês
| MEDLINE | ID: mdl-34853076
6.
Comparing Subjective Similarity of Automated Driving Styles to Objective Distance-Based Similarity.
Hum Factors
; 66(5): 1545-1563, 2024 May.
Artigo
em Inglês
| MEDLINE | ID: mdl-36602523
7.
Complement dysregulation in the central nervous system during development and disease.
Semin Immunol
; 45: 101340, 2019 10.
Artigo
em Inglês
| MEDLINE | ID: mdl-31708347
8.
Trust and trustworthy artificial intelligence: A research agenda for AI in the environmental sciences.
Risk Anal
; 2023 Nov 08.
Artigo
em Inglês
| MEDLINE | ID: mdl-37939398
9.
Trusting Automation: Designing for Responsivity and Resilience.
Hum Factors
; 65(1): 137-165, 2023 02.
Artigo
em Inglês
| MEDLINE | ID: mdl-33906505
10.
It's Not Only What You Say, But Also How You Say It: Machine Learning Approach to Estimate Trust from Conversation.
Hum Factors
; : 187208231166624, 2023 Apr 28.
Artigo
em Inglês
| MEDLINE | ID: mdl-37116009
11.
Attribution Errors by People and Intelligent Machines.
Hum Factors
; 65(7): 1293-1305, 2023 11.
Artigo
em Inglês
| MEDLINE | ID: mdl-34387108
12.
Glucose clearance and uptake is increased in the SOD1G93A mouse model of amyotrophic lateral sclerosis through an insulin-independent mechanism.
FASEB J
; 35(7): e21707, 2021 07.
Artigo
em Inglês
| MEDLINE | ID: mdl-34118098
13.
Absence of the C5a Receptor C5aR2 Worsens Ischemic Tissue Injury by Increasing C5aR1-Mediated Neutrophil Infiltration.
J Immunol
; 205(10): 2834-2839, 2020 11 15.
Artigo
em Inglês
| MEDLINE | ID: mdl-33028618
14.
A validated quantitative method for the assessment of neuroprotective barrier impairment in neurodegenerative disease models.
J Neurochem
; 158(3): 807-817, 2021 08.
Artigo
em Inglês
| MEDLINE | ID: mdl-32628780
15.
The potential interplay between energy metabolism and innate complement activation in amyotrophic lateral sclerosis.
FASEB J
; 34(6): 7225-7233, 2020 06.
Artigo
em Inglês
| MEDLINE | ID: mdl-32307753
16.
Clinical and electrophysiological examination of pinch strength in patients with amyotrophic lateral sclerosis.
Muscle Nerve
; 63(1): 108-113, 2021 01.
Artigo
em Inglês
| MEDLINE | ID: mdl-33118631
17.
The Complement Receptor C5aR2: A Powerful Modulator of Innate and Adaptive Immunity.
J Immunol
; 202(12): 3339-3348, 2019 06 15.
Artigo
em Inglês
| MEDLINE | ID: mdl-31160390
18.
Assessing Drivers' Trust of Automated Vehicle Driving Styles With a Two-Part Mixed Model of Intervention Tendency and Magnitude.
Hum Factors
; 63(2): 197-209, 2021 03.
Artigo
em Inglês
| MEDLINE | ID: mdl-31596618
19.
Designing for the Extremes: Modeling Drivers' Response Time to Take Back Control From Automation Using Bayesian Quantile Regression.
Hum Factors
; 63(3): 519-530, 2021 05.
Artigo
em Inglês
| MEDLINE | ID: mdl-31874049
20.
The microglial NLRP3 inflammasome is activated by amyotrophic lateral sclerosis proteins.
Glia
; 68(2): 407-421, 2020 02.
Artigo
em Inglês
| MEDLINE | ID: mdl-31596526