Tracing Differential Pathways of Risk: Associations Among Family Adversity, Cortisol, and Cognitive Functioning in Childhood.

Guided by family risk and allostasis theoretical frameworks, the present study utilized a prospective longitudinal design to examine associations among family risk experiences, basal cortisol patterns, and cognitive functioning in children. The sample included 201 low-income children living within a midsize city in the Northeastern United States. Children were assessed at ages 2, 3, and 4 years. Growth-mixture modeling analyses revealed three basal cortisol patterns (elevated, moderate, low) and these remained relatively stable across time. Exposure to greater levels of family instability and maternal unresponsiveness predicted elevated and low cortisol patterns, which were associated with lower child cognitive functioning at age 4. Findings have implications for family risk processes that may underlie risk-related disparities in child cognitive outcomes.

Interparental violence and childhood adjustment: how and why maternal sensitivity is a protective factor.

This study examined sensitive parenting as a protective factor in relations between interparental violence (IPV) and children's coping and psychological adjustment. Using a multimethod approach, a high-risk sample of 201 two-year-olds and their mothers participated in three annual waves of data collection. Moderator analyses revealed that sensitive parenting buffered the risk posed by IPV on children's changes in externalizing and prosocial development over a 2-year period. Tests of mediated moderation further indicated that sensitive parenting protected children from the vulnerability of growing up in a violent home through its association with lower levels of children's angry reactivity to interparental conflict. Results highlight the significance of identifying the mechanisms that mediate protective factors in models of family adversity.

Tracing the cascade of children's insecurity in the interparental relationship: the role of stage-salient tasks.

This study examined whether children's difficulties with stage-salient tasks served as an explanatory mechanism in the pathway between their insecurity in the interparental relationship and their disruptive behavior problems. Using a multimethod, multi-informant design, 201 two-year-old children and their mothers participated in 3 annual measurement occasions. Structural equation modeling analyses indicated that coder ratings of children's insecure responses to interparental conflict from a maternal interview predicted observer ratings of their difficulties with stage-salient tasks (i.e., emotion regulation, autonomy, resourceful problem solving) 1 year later after controlling for initial stage-salient task performance. Stage-salient task difficulties, in turn, predicted experimenter reports of children's behavior problems 1 year later. Associations remained robust in the broader context of other pathways hypothesized in prevailing developmental cascade models.

Pathways and processes of risk in associations among maternal antisocial personality symptoms, interparental aggression, and preschooler's psychopathology.

Two studies examined the nature and processes underlying the joint role of interparental aggression and maternal antisocial personality as predictors of children's disruptive behavior problems. Participants for both studies included a high-risk sample of 201 mothers and their 2-year-old children in a longitudinal, multimethod design. Addressing the form of the interplay between interparental aggression and maternal antisocial personality as risk factors for concurrent and prospective levels of child disruptive problems, the Study 1 findings indicated that maternal antisocial personality was a predictor of the initial levels of preschooler's disruptive problems independent of the effects of interparental violence, comorbid forms of maternal psychopathology, and socioeconomic factors. In attesting to the salience of interparental aggression in the lives of young children, latent difference score analyses further revealed that interparental aggression mediated the link between maternal antisocial personality and subsequent changes in child disruptive problems over a 1-year period. To identify the family mechanisms that account for the two forms of intergenerational transmission of disruptive problems identified in Study 1, Study 2 explored the role of children's difficult temperament, emotional reactivity to interparental conflict, adrenocortical reactivity in a challenging parent-child task, and experiences with maternal parenting as mediating processes. Analyses identified child emotional reactivity to conflict and maternal unresponsiveness as mediators in pathways between interparental aggression and preschooler's disruptive problems. The findings further supported the role of blunted adrenocortical reactivity as an allostatic mediator of the associations between parental unresponsiveness and child disruptive problems.

Children's patterns of emotional reactivity to conflict as explanatory mechanisms in links between interpartner aggression and child physiological functioning.

BACKGROUND: This paper examined children's fearful, sad, and angry reactivity to interparental conflict as mediators of associations between their exposure to interparental aggression and physiological functioning. METHODS: Participants included 200 toddlers and their mothers. Assessments of interparental aggression and children's emotional reactivity were derived from maternal surveys and a semi-structured interview. Cortisol levels and cardiac indices of sympathetic nervous system (SNS) and parasympathetic nervous system (PNS) activity were used to assess toddler physiological functioning. RESULTS: Results indicated that toddler exposure to interparental aggression was associated with greater cortisol levels and PNS activity and diminished SNS activity. Toddler angry emotional reactivity mediated associations between interparental aggression and cortisol and PNS functioning. Fearful emotional reactivity was a mediator of the link between interparental aggression and SNS functioning. CONCLUSIONS: The results are interpreted within conceptualizations of how exposure and reactivity to family risk organize individual differences in physiological functioning.

Interparental violence, maternal emotional unavailability and children's cortisol functioning in family contexts.

Our goal in the present study was to examine the specificity of pathways among interparental violence, maternal emotional unavailability, and children's cortisol reactivity to emotional stressors within interparental and parent-child relationships. The study also tested whether detrimental family contexts were associated, on average, with hypocortisolism or hypercortisolism responses to stressful family interactions in young children. Participants included 201 toddlers and their mothers who were from impoverished backgrounds and who experienced disproportionate levels of family violence. Assessments of interparental violence were derived from maternal surveys and interviews, whereas maternal emotional unavailability was assessed through maternal reports and observer ratings of caregiving. Salivary cortisol levels were sampled at 3 time points before and after laboratory paradigms designed to elicit children's reactivity to stressful interparental and parent-child contexts. Results indicated that interparental violence and the mother's emotional unavailability were differentially associated with children's adrenocorticol stress reactivity. Furthermore, these family risk contexts predicted lower cortisol change in response to distress. The results are interpreted in the context of risky family and emotional security theory conceptualizations that underscore how family contexts differentially impact children's physiological regulatory capacities.

Mother's parenting practices as explanatory mechanisms in associations between interparental violence and child adjustment.

This paper examined maternal parenting behaviors as mediators of associations between interparental violence and young children's internalizing and externalizing symptomatology. Participants included 201 toddlers and their mothers. Assessments of interparental violence and children's symptoms were derived from maternal surveys. Maternal parenting behaviors were assessed during an observational paradigm and coded for hostility, responsiveness and disengagement. Results indicated that mother's responsiveness and disengagement mediated associations between interparental violence and children's internalizing (e.g., withdrawn, inhibited, anxious, depressed behaviors) and externalizing (e.g., aggressive behaviors, attentional difficulties) symptoms. The results are interpreted in the context of conceptualizations that underscore how different dimensions of maternal parenting behaviors may play key explanatory roles in understanding associations between interparental violence and children's adjustment difficulties.