Detalhe da pesquisa
1.
Kidney-Specific CAP1/Prss8-Deficient Mice Maintain ENaC-Mediated Sodium Balance through an Aldosterone Independent Pathway.
Int J Mol Sci
; 23(12)2022 Jun 16.
Artigo
em Inglês
| MEDLINE | ID: mdl-35743186
2.
Plasma Potassium Determines NCC Abundance in Adult Kidney-Specific γENaC Knockout.
J Am Soc Nephrol
; 29(3): 977-990, 2018 03.
Artigo
em Inglês
| MEDLINE | ID: mdl-29371419
3.
Severe hyperkalemia is rescued by low-potassium diet in renal ßENaC-deficient mice.
Pflugers Arch
; 469(10): 1387-1399, 2017 10.
Artigo
em Inglês
| MEDLINE | ID: mdl-28567665
4.
Severe Salt-Losing Syndrome and Hyperkalemia Induced by Adult Nephron-Specific Knockout of the Epithelial Sodium Channel α-Subunit.
J Am Soc Nephrol
; 27(8): 2309-18, 2016 08.
Artigo
em Inglês
| MEDLINE | ID: mdl-26701978
5.
Adult nephron-specific MR-deficient mice develop a severe renal PHA-1 phenotype.
Pflugers Arch
; 468(5): 895-908, 2016 05.
Artigo
em Inglês
| MEDLINE | ID: mdl-26762397
6.
The CAP1/Prss8 catalytic triad is not involved in PAR2 activation and protease nexin-1 (PN-1) inhibition.
FASEB J
; 28(11): 4792-805, 2014 Nov.
Artigo
em Inglês
| MEDLINE | ID: mdl-25138159
7.
Colon-specific deletion of epithelial sodium channel causes sodium loss and aldosterone resistance.
J Am Soc Nephrol
; 25(7): 1453-64, 2014 Jul.
Artigo
em Inglês
| MEDLINE | ID: mdl-24480829
8.
Mutations of the serine protease CAP1/Prss8 lead to reduced embryonic viability, skin defects, and decreased ENaC activity.
Am J Pathol
; 181(2): 605-15, 2012 Aug.
Artigo
em Inglês
| MEDLINE | ID: mdl-22705055
9.
Deletion of the serine protease CAP2/Tmprss4 leads to dysregulated renal water handling upon dietary potassium depletion.
Sci Rep
; 9(1): 19540, 2019 12 20.
Artigo
em Inglês
| MEDLINE | ID: mdl-31863073
10.
Lack of Renal Tubular Glucocorticoid Receptor Decreases the Thiazide-Sensitive Na+/Cl- Cotransporter NCC and Transiently Affects Sodium Handling.
Front Physiol
; 10: 989, 2019.
Artigo
em Inglês
| MEDLINE | ID: mdl-31474871