Detalhe da pesquisa
1.
Targeted deubiquitination rescues distinct trafficking-deficient ion channelopathies.
Nat Methods
; 17(12): 1245-1253, 2020 12.
Artigo
em Inglês
| MEDLINE | ID: mdl-33169015
2.
Engineering selectivity into RGK GTPase inhibition of voltage-dependent calcium channels.
Proc Natl Acad Sci U S A
; 115(47): 12051-12056, 2018 11 20.
Artigo
em Inglês
| MEDLINE | ID: mdl-30397133
3.
The human Cx26-D50A and Cx26-A88V mutations causing keratitis-ichthyosis-deafness syndrome display increased hemichannel activity.
Am J Physiol Cell Physiol
; 304(12): C1150-8, 2013 Jun 15.
Artigo
em Inglês
| MEDLINE | ID: mdl-23447037
4.
Cardiac CaV1.2 channels require ß subunits for ß-adrenergic-mediated modulation but not trafficking.
J Clin Invest
; 129(2): 647-658, 2019 02 01.
Artigo
em Inglês
| MEDLINE | ID: mdl-30422117
5.
Targeting the CaVα-CaVß interaction yields an antagonist of the N-type CaV2.2 channel with broad antinociceptive efficacy.
Pain
; 160(7): 1644-1661, 2019 07.
Artigo
em Inglês
| MEDLINE | ID: mdl-30933958
6.
Regulation of microdomain voltage-gated L-type calcium channels in cardiac health and disease.
Curr Opin Physiol
; 2: 13-18, 2018 Apr.
Artigo
em Inglês
| MEDLINE | ID: mdl-29963649
7.
Connexin26 Mutations Causing Palmoplantar Keratoderma and Deafness Interact with Connexin43, Modifying Gap Junction and Hemichannel Properties.
J Invest Dermatol
; 136(1): 225-235, 2016 Jan.
Artigo
em Inglês
| MEDLINE | ID: mdl-26763442