Detalhe da pesquisa
1.
Macrophage Migration Inhibitory Factor (MIF) Makes Complex Contributions to Pain-Related Hyperactivity of Nociceptors after Spinal Cord Injury.
J Neurosci
; 2022 May 23.
Artigo
em Inglês
| MEDLINE | ID: mdl-35610050
2.
Exaptation and Evolutionary Adaptation in Nociceptor Mechanisms Driving Persistent Pain.
Brain Behav Evol
; 98(6): 314-330, 2023.
Artigo
em Inglês
| MEDLINE | ID: mdl-38035556
3.
Depolarization-Dependent C-Raf Signaling Promotes Hyperexcitability and Reduces Opioid Sensitivity of Isolated Nociceptors after Spinal Cord Injury.
J Neurosci
; 40(34): 6522-6535, 2020 08 19.
Artigo
em Inglês
| MEDLINE | ID: mdl-32690613
4.
Oral Dimethyl Fumarate Reduces Peripheral Neuropathic Pain in Rodents via NFE2L2 Antioxidant Signaling.
Anesthesiology
; 132(2): 343-356, 2020 02.
Artigo
em Inglês
| MEDLINE | ID: mdl-31939850
5.
Zinc Inhibits TRPV1 to Alleviate Chemotherapy-Induced Neuropathic Pain.
J Neurosci
; 38(2): 474-483, 2018 01 10.
Artigo
em Inglês
| MEDLINE | ID: mdl-29192128
6.
Persistent Electrical Activity in Primary Nociceptors after Spinal Cord Injury Is Maintained by Scaffolded Adenylyl Cyclase and Protein Kinase A and Is Associated with Altered Adenylyl Cyclase Regulation.
J Neurosci
; 36(5): 1660-8, 2016 Feb 03.
Artigo
em Inglês
| MEDLINE | ID: mdl-26843647
7.
Molecular and cellular mechanisms that initiate pain and itch.
Cell Mol Life Sci
; 72(17): 3201-23, 2015 Sep.
Artigo
em Inglês
| MEDLINE | ID: mdl-25894692
8.
Persistent pain after spinal cord injury is maintained by primary afferent activity.
J Neurosci
; 34(32): 10765-9, 2014 Aug 06.
Artigo
em Inglês
| MEDLINE | ID: mdl-25100607
9.
Squid have nociceptors that display widespread long-term sensitization and spontaneous activity after bodily injury.
J Neurosci
; 33(24): 10021-6, 2013 Jun 12.
Artigo
em Inglês
| MEDLINE | ID: mdl-23761897
10.
Readiness of nociceptor cell bodies to generate spontaneous activity results from background activity of diverse ion channels and high input resistance.
Pain
; 165(4): 893-907, 2024 Apr 01.
Artigo
em Inglês
| MEDLINE | ID: mdl-37862056
11.
Widespread latent hyperactivity of nociceptors outlasts enhanced avoidance behavior following incision injury.
bioRxiv
; 2024 Jan 31.
Artigo
em Inglês
| MEDLINE | ID: mdl-38352319
12.
Targeting Pain-evoking Transient Receptor Potential Channels for the Treatment of Pain.
Curr Neuropharmacol
; 11(6): 652-63, 2013 Dec.
Artigo
em Inglês
| MEDLINE | ID: mdl-24396340
13.
Persistent nociceptor hyperactivity as a painful evolutionary adaptation.
Trends Neurosci
; 46(3): 211-227, 2023 03.
Artigo
em Inglês
| MEDLINE | ID: mdl-36610893
14.
Major Differences in Transcriptional Alterations in Dorsal Root Ganglia Between Spinal Cord Injury and Peripheral Neuropathic Pain Models.
J Neurotrauma
; 40(9-10): 883-900, 2023 05.
Artigo
em Inglês
| MEDLINE | ID: mdl-36178348
15.
Electrophysiological Alterations Driving Pain-Associated Spontaneous Activity in Human Sensory Neuron Somata Parallel Alterations Described in Spontaneously Active Rodent Nociceptors.
J Pain
; 23(8): 1343-1357, 2022 08.
Artigo
em Inglês
| MEDLINE | ID: mdl-35292377
16.
Chronic spontaneous activity generated in the somata of primary nociceptors is associated with pain-related behavior after spinal cord injury.
J Neurosci
; 30(44): 14870-82, 2010 Nov 03.
Artigo
em Inglês
| MEDLINE | ID: mdl-21048146
17.
Peripheral injury induces long-term sensitization of defensive responses to visual and tactile stimuli in the squid Loligo pealeii, Lesueur 1821.
J Exp Biol
; 214(Pt 19): 3173-85, 2011 Oct 01.
Artigo
em Inglês
| MEDLINE | ID: mdl-21900465
18.
Erratum to: Molecular and cellular mechanisms that initiate pain and itch.
Cell Mol Life Sci
; 72(18): 3587-8, 2015 Sep.
Artigo
em Inglês
| MEDLINE | ID: mdl-26135056
19.
Serotonin enhances depolarizing spontaneous fluctuations, excitability, and ongoing activity in isolated rat DRG neurons via 5-HT4 receptors and cAMP-dependent mechanisms.
Neuropharmacology
; 184: 108408, 2021 02 15.
Artigo
em Inglês
| MEDLINE | ID: mdl-33220305
20.
Long-lasting synaptic potentiation induced by depolarization under conditions that eliminate detectable Ca2+ signals.
J Neurophysiol
; 103(3): 1283-94, 2010 Mar.
Artigo
em Inglês
| MEDLINE | ID: mdl-20042699