Detalhe da pesquisa
1.
The potential health risks of N,N-dimethylformamide: An updated review.
J Appl Toxicol
; 2024 Feb 10.
Artigo
em Inglês
| MEDLINE | ID: mdl-38339870
2.
N,N-dimethylformamide-induced acute liver damage is driven by the activation of NLRP3 inflammasome in liver macrophages of mice.
Ecotoxicol Environ Saf
; 238: 113609, 2022 Jun 15.
Artigo
em Inglês
| MEDLINE | ID: mdl-35551047
3.
Targeting macrophage polarization by Nrf2 agonists for treating various xenobiotics-induced toxic responses.
Toxicol Mech Methods
; 31(5): 334-342, 2021 Jun.
Artigo
em Inglês
| MEDLINE | ID: mdl-33627030
4.
NLRP3 inflammasome activation triggers severe inflammatory liver injury in N, N-dimethylformamide-exposed mice.
Sci Total Environ
; 929: 172653, 2024 Jun 15.
Artigo
em Inglês
| MEDLINE | ID: mdl-38649053
5.
Allyl methyl disulfide (AMDS) prevents N,N-dimethyl formamide-induced liver damage by suppressing oxidative stress and NLRP3 inflammasome activation.
Food Chem Toxicol
; 182: 114198, 2023 Dec.
Artigo
em Inglês
| MEDLINE | ID: mdl-37995826
6.
Diallyl disulfide suppresses the lipopolysaccharide-driven inflammatory response of macrophages by activating the Nrf2 pathway.
Food Chem Toxicol
; 159: 112760, 2022 Jan.
Artigo
em Inglês
| MEDLINE | ID: mdl-34896185
7.
Transformed ALDH2-/- hepatocytes by ethanol could serve as a useful tool for studying alcoholic hepatocarcinogenesis.
Med Hypotheses
; 146: 110366, 2021 Jan.
Artigo
em Inglês
| MEDLINE | ID: mdl-33208242
8.
Roles of peroxisome proliferator-activated receptor α in the pathogenesis of ethanol-induced liver disease.
Chem Biol Interact
; 327: 109176, 2020 Aug 25.
Artigo
em Inglês
| MEDLINE | ID: mdl-32534989