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Inhibition of CaMKII does not attenuate cardiac hypertrophy in mice with dysfunctional ryanodine receptor.
Chakraborty, Asima; Pasek, Daniel A; Huang, Tai-Qin; Gomez, Angela C; Yamaguchi, Naohiro; Anderson, Mark E; Meissner, Gerhard.
Afiliação
  • Chakraborty A; Department of Biochemistry and Biophysics, University of North Carolina, Chapel Hill, NC, United States of America.
  • Pasek DA; Department of Biochemistry and Biophysics, University of North Carolina, Chapel Hill, NC, United States of America.
  • Huang TQ; Department of Biochemistry and Biophysics, University of North Carolina, Chapel Hill, NC, United States of America.
  • Gomez AC; Department of Regenerative Medicine and Cell Biology, Medical University of South Carolina, Charleston, SC, United States of America.
  • Yamaguchi N; Department of Regenerative Medicine and Cell Biology, Medical University of South Carolina, Charleston, SC, United States of America.
  • Anderson ME; Division of Cardiovascular Medicine, Departments of Internal Medicine, and Molecular Physiology and Biophysics, University of Iowa, Iowa City, IA, United States of America.
  • Meissner G; Department of Biochemistry and Biophysics, University of North Carolina, Chapel Hill, NC, United States of America.
PLoS One ; 9(8): e104338, 2014.
Article em En | MEDLINE | ID: mdl-25093823

Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Cardiomegalia / Canal de Liberação de Cálcio do Receptor de Rianodina / Proteína Quinase Tipo 2 Dependente de Cálcio-Calmodulina Tipo de estudo: Diagnostic_studies / Prognostic_studies Limite: Animals Idioma: En Revista: PLoS One Assunto da revista: CIENCIA / MEDICINA Ano de publicação: 2014 Tipo de documento: Article País de afiliação: Estados Unidos

Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Cardiomegalia / Canal de Liberação de Cálcio do Receptor de Rianodina / Proteína Quinase Tipo 2 Dependente de Cálcio-Calmodulina Tipo de estudo: Diagnostic_studies / Prognostic_studies Limite: Animals Idioma: En Revista: PLoS One Assunto da revista: CIENCIA / MEDICINA Ano de publicação: 2014 Tipo de documento: Article País de afiliação: Estados Unidos