Increasing Notch signaling antagonizes PRC2-mediated silencing to promote reprograming of germ cells into neurons.

Seelk, Stefanie; Adrian-Kalchhauser, Irene; Hargitai, Balázs; Hajduskova, Martina; Gutnik, Silvia; Tursun, Baris; Ciosk, Rafal

Seelk, Stefanie; Adrian-Kalchhauser, Irene; Hargitai, Balázs; Hajduskova, Martina; Gutnik, Silvia; Tursun, Baris; Ciosk, Rafal.

Afiliação

Seelk S; Berlin Institute for Medical Systems Biology, Max-Delbrück-Center for Molecular Medicine, Berlin, Germany.
Adrian-Kalchhauser I; Friedrich Miescher Institute for Biomedical Research, Basel, Switzerland.
Hargitai B; Friedrich Miescher Institute for Biomedical Research, Basel, Switzerland.
Hajduskova M; Berlin Institute for Medical Systems Biology, Max-Delbrück-Center for Molecular Medicine, Berlin, Germany.
Gutnik S; Friedrich Miescher Institute for Biomedical Research, Basel, Switzerland.
Tursun B; Berlin Institute for Medical Systems Biology, Max-Delbrück-Center for Molecular Medicine, Berlin, Germany.
Ciosk R; Friedrich Miescher Institute for Biomedical Research, Basel, Switzerland.

Elife ; 52016 Sep 07.

Article em En | MEDLINE | ID: mdl-27602485

Palavras-chave

C. elegans; Notch; PRC2; T-ALL; developmental biology; germ cell; glp-1; leukemia; mes-2; mes-3; mes-6; reprograming; stem cell; stem cells; utx-1

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Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Tipo de estudo: Prognostic_studies Idioma: En Revista: Elife Ano de publicação: 2016 Tipo de documento: Article País de afiliação: Alemanha

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