Group B Streptococci Induce Proinflammatory Responses via a Protein Kinase D1-Dependent Pathway.

Upadhyay, Kirtikumar; Park, Jeoung-Eun; Yoon, Tae Won; Halder, Priyanka; Kim, Young-In; Metcalfe, Victoria; Talati, Ajay J; English, B Keith; Yi, Ae-Kyung

Upadhyay, Kirtikumar; Park, Jeoung-Eun; Yoon, Tae Won; Halder, Priyanka; Kim, Young-In; Metcalfe, Victoria; Talati, Ajay J; English, B Keith; Yi, Ae-Kyung.

Afiliação

Upadhyay K; Department of Pediatrics, The University of Tennessee Health Science Center, Memphis, TN 38163.
Park JE; Department of Obstetrics and Gynecology, The University of Tennessee Health Science Center, Memphis, TN 38163.
Yoon TW; Children's Foundation Research Institute, Le Bonheur Children's Hospital, Memphis, TN 38103.
Halder P; Department of Pediatrics, The University of Tennessee Health Science Center, Memphis, TN 38163.
Kim YI; Department of Microbiology, Immunology and Biochemistry, The University of Tennessee Health Science Center, Memphis, TN 38163; and.
Metcalfe V; Department of Microbiology, Immunology and Biochemistry, The University of Tennessee Health Science Center, Memphis, TN 38163; and.
Talati AJ; Department of Pediatrics, The University of Tennessee Health Science Center, Memphis, TN 38163.
English BK; Children's Foundation Research Institute, Le Bonheur Children's Hospital, Memphis, TN 38103.
Yi AK; Department of Microbiology, Immunology and Biochemistry, The University of Tennessee Health Science Center, Memphis, TN 38163; and.

J Immunol ; 198(11): 4448-4457, 2017 06 01.

Article em En | MEDLINE | ID: mdl-28461572

Assuntos

Inflamação/imunologia; Proteína Quinase C/metabolismo; Infecções Estreptocócicas/imunologia; Infecções Estreptocócicas/metabolismo; Streptococcus agalactiae/imunologia; Animais; Humanos; Recém-Nascido; Proteína Antagonista do Receptor de Interleucina 1/imunologia; Proteína Antagonista do Receptor de Interleucina 1/metabolismo; Macrófagos/imunologia; Macrófagos/microbiologia; Camundongos; Fator 88 de Diferenciação Mieloide; NF-kappa B/metabolismo; Proteína Quinase C/antagonistas & inibidores; Proteína Quinase C/deficiência; Sepse/microbiologia; Transdução de Sinais; Fator de Necrose Tumoral alfa/biossíntese

Texto completo

Imprimir

XML

PubMed Links

Buscar no Google

Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Infecções Estreptocócicas / Streptococcus agalactiae / Proteína Quinase C / Inflamação Limite: Animals / Humans / Newborn Idioma: En Revista: J Immunol Ano de publicação: 2017 Tipo de documento: Article

Texto completo

Imprimir

XML

PubMed Links

Buscar no Google