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Calreticulin promotes EGF-induced EMT in pancreatic cancer cells via Integrin/EGFR-ERK/MAPK signaling pathway.
Sheng, Weiwei; Chen, Chuanping; Dong, Ming; Wang, Guosen; Zhou, Jianping; Song, He; Li, Yang; Zhang, Jian; Ding, Shuangning.
Afiliação
  • Sheng W; Department of Gastrointestinal Surgery, the First Hospital, China Medical University, Shenyang 110001, China.
  • Chen C; Department of Clinical Laboratory, the Sixth Peoples' hospital of Shenyang, Shenyang 110003, China.
  • Dong M; Department of Gastrointestinal Surgery, the First Hospital, China Medical University, Shenyang 110001, China.
  • Wang G; Department of Gastrointestinal Surgery, the First Hospital, China Medical University, Shenyang 110001, China.
  • Zhou J; Department of Gastrointestinal Surgery, the First Hospital, China Medical University, Shenyang 110001, China.
  • Song H; Department of Gastrointestinal Surgery, the First Hospital, China Medical University, Shenyang 110001, China.
  • Li Y; Department of Cell Biology, China Medical University, Shenyang 110013, China.
  • Zhang J; Department of Cell Biology, China Medical University, Shenyang 110013, China.
  • Ding S; Department of Endocrinology and Metabolism in Liaoning Province, the First Hospital of China Medical University, Shenyang 110001, China.
Cell Death Dis ; 8(10): e3147, 2017 10 26.
Article em En | MEDLINE | ID: mdl-29072694

Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Neoplasias Pancreáticas / Integrinas / Sistema de Sinalização das MAP Quinases / Carcinoma Ductal Pancreático / Calreticulina / Fator de Crescimento Epidérmico Tipo de estudo: Prognostic_studies Limite: Animals / Humans Idioma: En Revista: Cell Death Dis Ano de publicação: 2017 Tipo de documento: Article País de afiliação: China

Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Neoplasias Pancreáticas / Integrinas / Sistema de Sinalização das MAP Quinases / Carcinoma Ductal Pancreático / Calreticulina / Fator de Crescimento Epidérmico Tipo de estudo: Prognostic_studies Limite: Animals / Humans Idioma: En Revista: Cell Death Dis Ano de publicação: 2017 Tipo de documento: Article País de afiliação: China