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Endothelial deletion of Ino80 disrupts coronary angiogenesis and causes congenital heart disease.
Rhee, Siyeon; Chung, Jae I; King, Devin A; D'amato, Gaetano; Paik, David T; Duan, Anna; Chang, Andrew; Nagelberg, Danielle; Sharma, Bikram; Jeong, Youngtae; Diehn, Maximilian; Wu, Joseph C; Morrison, Ashby J; Red-Horse, Kristy.
Afiliação
  • Rhee S; Department of Biology, Stanford University, 371 Serra Mall, Stanford, CA, 94305, USA.
  • Chung JI; Department of Biology, Stanford University, 371 Serra Mall, Stanford, CA, 94305, USA.
  • King DA; Department of Biology, Stanford University, 371 Serra Mall, Stanford, CA, 94305, USA.
  • D'amato G; Department of Biology, Stanford University, 371 Serra Mall, Stanford, CA, 94305, USA.
  • Paik DT; Stanford Cardiovascular Institute, Stanford University School of Medicine, Stanford, CA, 94305, USA.
  • Duan A; Department of Medicine, Stanford University School of Medicine, Stanford, CA, 94305, USA.
  • Chang A; Institute for Stem Cell Biology and Regenerative Medicine, Stanford University School of Medicine, Stanford, CA, 94305, USA.
  • Nagelberg D; Department of Biology, Stanford University, 371 Serra Mall, Stanford, CA, 94305, USA.
  • Sharma B; Department of Biology, Stanford University, 371 Serra Mall, Stanford, CA, 94305, USA.
  • Jeong Y; Department of Biology, Stanford University, 371 Serra Mall, Stanford, CA, 94305, USA.
  • Diehn M; Department of Biology, Stanford University, 371 Serra Mall, Stanford, CA, 94305, USA.
  • Wu JC; Institute for Stem Cell Biology and Regenerative Medicine, Stanford University School of Medicine, Stanford, CA, 94305, USA.
  • Morrison AJ; Stanford Cancer Institute, Stanford University School of Medicine, Stanford, CA, 94305, USA.
  • Red-Horse K; Department of Radiation Oncology, Stanford University School of Medicine, Stanford, CA, 94305, USA.
Nat Commun ; 9(1): 368, 2018 01 25.
Article em En | MEDLINE | ID: mdl-29371594
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Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Endotélio Vascular / Adenosina Trifosfatases / Cardiopatias Congênitas / Neovascularização Patológica Tipo de estudo: Etiology_studies Limite: Animals / Humans Idioma: En Revista: Nat Commun Assunto da revista: BIOLOGIA / CIENCIA Ano de publicação: 2018 Tipo de documento: Article País de afiliação: Estados Unidos
Texto completo
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XML
PubMed Links
Buscar no Google

Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Endotélio Vascular / Adenosina Trifosfatases / Cardiopatias Congênitas / Neovascularização Patológica Tipo de estudo: Etiology_studies Limite: Animals / Humans Idioma: En Revista: Nat Commun Assunto da revista: BIOLOGIA / CIENCIA Ano de publicação: 2018 Tipo de documento: Article País de afiliação: Estados Unidos