ERß alters the chemosensitivity of luminal breast cancer cells by regulating p53 function.

Bado, Igor; Pham, Eric; Soibam, Benjamin; Nikolos, Fotis; Gustafsson, Jan-Åke; Thomas, Christoforos

Bado, Igor; Pham, Eric; Soibam, Benjamin; Nikolos, Fotis; Gustafsson, Jan-Åke; Thomas, Christoforos.

Afiliação

Bado I; Department of Biology and Biochemistry, Center for Nuclear Receptors and Cell Signaling, University of Houston, Houston, Texas, USA.
Pham E; Department of Biology and Biochemistry, University of Houston, Houston, Texas, USA.
Soibam B; Department of Computer Science and Engineering Technology, University of Houston-Downtown, Huston, Texas, USA.
Nikolos F; Department of Biology and Biochemistry, Center for Nuclear Receptors and Cell Signaling, University of Houston, Houston, Texas, USA.
Gustafsson JÅ; Department of Biology and Biochemistry, Center for Nuclear Receptors and Cell Signaling, University of Houston, Houston, Texas, USA.
Thomas C; Center for Innovative Medicine, Department of Biosciences and Nutrition, Karolinska Institutet, Huddinge, Sweden.

Oncotarget ; 9(32): 22509-22522, 2018 Apr 27.

Article em En | MEDLINE | ID: mdl-29854295

Palavras-chave

DNA damage response; breast cancer; estrogen receptor ß; p53; therapy response

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Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Idioma: En Revista: Oncotarget Ano de publicação: 2018 Tipo de documento: Article País de afiliação: Estados Unidos

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