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Addendum: TGF-ß signaling alters H4K20me3 status via miR-29 and contributes to cellular senescence and cardiac aging.
Lyu, Guoliang; Guan, Yiting; Zhang, Chao; Zong, Le; Sun, Lei; Huang, Xiaoke; Huang, Li; Zhang, Lijun; Tian, Xiao-Li; Zhou, Zhongjun; Tao, Wei.
Afiliação
  • Lyu G; The MOE Key Laboratory of Cell Proliferation and Differentiation, School of Life Sciences, Peking University, Beijing, 100871, China.
  • Guan Y; The MOE Key Laboratory of Cell Proliferation and Differentiation, School of Life Sciences, Peking University, Beijing, 100871, China.
  • Zhang C; The MOE Key Laboratory of Cell Proliferation and Differentiation, School of Life Sciences, Peking University, Beijing, 100871, China.
  • Zong L; The MOE Key Laboratory of Cell Proliferation and Differentiation, School of Life Sciences, Peking University, Beijing, 100871, China.
  • Sun L; The MOE Key Laboratory of Cell Proliferation and Differentiation, School of Life Sciences, Peking University, Beijing, 100871, China.
  • Huang X; The MOE Key Laboratory of Cell Proliferation and Differentiation, School of Life Sciences, Peking University, Beijing, 100871, China.
  • Huang L; The MOE Key Laboratory of Cell Proliferation and Differentiation, School of Life Sciences, Peking University, Beijing, 100871, China.
  • Zhang L; The MOE Key Laboratory of Cell Proliferation and Differentiation, School of Life Sciences, Peking University, Beijing, 100871, China.
  • Tian XL; Department of Human Population Genetics, Human Aging Research Institute and School of Life Science, Nanchang University, Nanchang, 330031, China.
  • Zhou Z; School of Biomedical Sciences, LKS Faculty of Medicine, The University of Hong Kong, 21 Sassoon Road, Hong Kong, China.
  • Tao W; Shenzhen Institute of Innovation and Research, The University of Hong Kong, Nanshan, Shenzhen 518000, China.
Nat Commun ; 9(1): 4134, 2018 10 08.
Article em En | MEDLINE | ID: mdl-30297907

Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Idioma: En Revista: Nat Commun Assunto da revista: BIOLOGIA / CIENCIA Ano de publicação: 2018 Tipo de documento: Article País de afiliação: China

Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Idioma: En Revista: Nat Commun Assunto da revista: BIOLOGIA / CIENCIA Ano de publicação: 2018 Tipo de documento: Article País de afiliação: China