Polymorphic Membrane Protein 17G of <i>Chlamydia psittaci</i> Mediated the Binding and Invasion of Bacteria to Host Cells by Interacting and Activating EGFR of the Host.

Li, Xiaohui; Zuo, Zonghui; Wang, Yihui; Hegemann, Johannes H; He, Cheng

Polymorphic Membrane Protein 17G of Chlamydia psittaci Mediated the Binding and Invasion of Bacteria to Host Cells by Interacting and Activating EGFR of the Host.

Li, Xiaohui; Zuo, Zonghui; Wang, Yihui; Hegemann, Johannes H; He, Cheng.

Afiliação

Li X; Key Lab of Animal Epidemiology and Zoonoses of Ministry of Agriculture and Rural Affairs, College of Veterinary Medicine, China Agricultural University, Beijing, China.
Zuo Z; Tianjin Key Laboratory of Agricultural Animal Breeding and Healthy Husbandry, College of Animal Science and Veterinary Medicine, Tianjin Agricultural University, Tianjin, China.
Wang Y; Department of Biology, Institute for Functional Microbial Genomics, Heinrich-Heine-Universität Düsseldorf, Düsseldorf, Germany.
Hegemann JH; Key Lab of Animal Epidemiology and Zoonoses of Ministry of Agriculture and Rural Affairs, College of Veterinary Medicine, China Agricultural University, Beijing, China.
He C; Department of Biology, Institute for Functional Microbial Genomics, Heinrich-Heine-Universität Düsseldorf, Düsseldorf, Germany.

Front Immunol ; 12: 818487, 2021.

Article em En | MEDLINE | ID: mdl-35173712

Assuntos

Chlamydophila psittaci/fisiologia; Receptores ErbB/metabolismo; Interações Hospedeiro-Patógeno; Proteínas de Membrana/metabolismo; Psitacose/metabolismo; Psitacose/microbiologia; Adesão Celular; Linhagem Celular; Receptores ErbB/agonistas; Humanos; Proteínas de Membrana/antagonistas & inibidores; Proteínas de Membrana/genética; Fosforilação; Ligação Proteica

Palavras-chave

C. psittaci; EGFR; Pmp17G; adhesion; invasion

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Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Psitacose / Chlamydophila psittaci / Interações Hospedeiro-Patógeno / Receptores ErbB / Proteínas de Membrana Tipo de estudo: Prognostic_studies Limite: Humans Idioma: En Revista: Front Immunol Ano de publicação: 2021 Tipo de documento: Article País de afiliação: China

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