Interleukin-35 expression promotes hepatocellular carcinogenesis by inducing Î³Î´ T-cell exhaustion.

Ren, He; Liu, Xin; Xu, Qiao; Li, Wanjing; Zhao, Na

Ren, He; Liu, Xin; Xu, Qiao; Li, Wanjing; Zhao, Na.

Afiliação

Ren H; Department of Breast Surgery, Harbin Medical University Cancer Hospital, Harbin 150081, China; Department of General Surgery, Tianjin Medical University General Hospital, Tianjin Medical University, Tianjin 300052, China.
Liu X; Department of General Surgery, Tianjin Medical University General Hospital, Tianjin Medical University, Tianjin 300052, China.
Xu Q; Department of General Surgery, Tianjin Medical University General Hospital, Tianjin Medical University, Tianjin 300052, China.
Li W; Department of General Surgery, Tianjin Medical University General Hospital, Tianjin Medical University, Tianjin 300052, China.
Zhao N; Department of General Surgery, Tianjin Medical University General Hospital, Tianjin Medical University, Tianjin 300052, China. Electronic address: nasy3367@126.com.

Genomics ; 115(3): 110639, 2023 05.

Article em En | MEDLINE | ID: mdl-37187255

Assuntos

Carcinoma Hepatocelular; Neoplasias Hepáticas; Humanos; Linfócitos T; Carcinoma Hepatocelular/metabolismo; Neoplasias Hepáticas/metabolismo; Exaustão das Células T; Interleucinas; Carcinogênese; Microambiente Tumoral

Palavras-chave

Hepatocellular carcinoma; Interleukin-35; T-cell exhaustion; Tumor microenvironment; Î³Î´ T-cell

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Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Carcinoma Hepatocelular / Neoplasias Hepáticas Tipo de estudo: Prognostic_studies Limite: Humans Idioma: En Revista: Genomics Assunto da revista: GENETICA Ano de publicação: 2023 Tipo de documento: Article País de afiliação: China

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