Cmah deficiency blunts cellular senescence in adipose tissues and improves whole-body glucose metabolism in aged mice.

Takeda, Reo; Tabuchi, Ayaka; Nonaka, Yudai; Kano, Ryotaro; Sudo, Mizuki; Kano, Yutaka; Hoshino, Daisuke

Takeda, Reo; Tabuchi, Ayaka; Nonaka, Yudai; Kano, Ryotaro; Sudo, Mizuki; Kano, Yutaka; Hoshino, Daisuke.

Afiliação

Takeda R; Department of Engineering Science, The University of Electro-communications, Tokyo, Japan.
Tabuchi A; Japan Society for the Promotion of Science, Tokyo, Japan.
Nonaka Y; Department of Engineering Science, The University of Electro-communications, Tokyo, Japan.
Kano R; Institute of Liberal Arts and Sciences, Kanazawa University, Ishikawa, Japan.
Sudo M; Department of Engineering Science, The University of Electro-communications, Tokyo, Japan.
Kano Y; Japan Society for the Promotion of Science, Tokyo, Japan.
Hoshino D; Physical Fitness Research Institute, Meiji Yasuda Life Foundation of Health and Welfare, Tokyo, Japan.

Geriatr Gerontol Int ; 23(12): 958-964, 2023 Dec.

Article em En | MEDLINE | ID: mdl-37968438

Assuntos

Insulinas; Ácido N-Acetilneuramínico; Animais; Camundongos; Senescência Celular; Glucose; Camundongos Knockout; Ácido N-Acetilneuramínico/metabolismo

Palavras-chave

Cmah; adipose tissue; aging; cellular senescence; glucose metabolism

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Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Ácido N-Acetilneuramínico / Insulinas Limite: Animals Idioma: En Revista: Geriatr Gerontol Int Ano de publicação: 2023 Tipo de documento: Article País de afiliação: Japão

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