Upper-body kinematics in team-handball throw, tennis serve, and volleyball spike.

Overarm movements are essential skills in many different sport games; however, the adaptations to different sports are not well understood. The aim of the study was to analyze upper-body kinematics in the team-handball throw, tennis serve, and volleyball spike, and to calculate differences in the proximal-to-distal sequencing and joint movements. Three-dimensional kinematic data were analyzed via the Vicon motion capturing system. The subjects (elite players) were instructed to perform a team-handball jump throw, tennis serve, and volleyball spike with a maximal ball velocity and to hit a specific target. Significant differences (P < 0.05) between the three overarm movements were found in 17 of 24 variables. The order of the proximal-to-distal sequencing was equal in the three analyzed overarm movements. Equal order of the proximal-to-distal sequencing and similar angles in the acceleration phase suggest there is a general motor pattern in overarm movements. However, overarm movements appear to be modifiable in situations such as for throwing or hitting a ball with or without a racket, and due to differences at takeoff (with one or two legs).

Interactions between adipose, bone, and muscle tissue markers during acute negative energy balance in male rowers.

AIM: The purpose of this investigation was to study the influence of prolonged low-intensity single scull rowing exercise on plasma adipocytokine and osteokine concentrations in trained male rowers. Nine single scull rowers (age: 20.1±1.6 yrs; height: 184.1±4.6 cm; body mass: 81.2±5.3 kg; %body fat: 11.1±3.9) participated in this study. METHODS: Venous blood samples were obtained before and after a ~2 h constant load on-water sculling training session (distance: 20.6±1.5 km; HR: 133±4 bpm; intensity: 80.3±1.7% of the HR turn-point). RESULTS: The training session created an energy deficit of 1 200-1 500 kcal. Plasma adiponectin increased (+12.2%; P<0.05) while leptin decreased (-23.1%; P<0.05) at 30 min postexercise. Plasma osteocalcin (+23.7%; P<0.05) and type I carboxyterminal telopeptide (ICTP) (+28.6%; P<0.05) were increased on completion of the training session. Plasma IL-6 concentration was increased (P<0.05) about four-fold while insulin concentration was decreased (P<0.05) more than three-fold upon completion of exercise. There were no differences in TNF-α, glucose, testosterone and cortisol concentrations over time. Plasma adiponectin (r=0.59) and osteocalcin (r=0.57) concentrations measured immediately after the training session were related (P<0.05) to the distance covered. CONCLUSION: Acute negative energy balance induced by a single endurance rowing training session elicited an inverse metabolic response in adiponectin and osteocalcin concentrations in male rowers. Our results suggest that peripheral markers of negative energy balance, such as adiponectin and osteocalcin, may serve as signals for metabolic reaction to the energy cost of acute exercise in athletes.

[Complications during exercise testing]. / Komplikationen bei der Ergometrie.

Exercise electrocardiogram forms the basis for diagnosis of ischemia in coronary heart disease. Blood pressure behavior, physical fitness, training heart rate and possible cardiac arrhythmias can additionally be assessed using bicycle ergometry or treadmill testing. When the indications for and contraindications to exercise testing (either bicycle ergometry or treadmill testing) are closely observed, serious complications are rare. However, it is important that the treating physician is aware of and able to recognize possible complications. The present article discusses possible cardiovascular complications and their incidence.

Kinematic analysis of volleyball spike jump.

The purpose of this study was to determine the influence of upper and lower extremity movements on the volleyball spike jump (SJ) and how this movement may differ from the standing vertical jumps due to its asymmetry. The 3-D kinematics of body segments were measured in 16 experienced volleyball players with a VICON motion capture system. The jump heights (JH) of counter-movement (CM) and SJ were determined utilizing a force platform. A significant correlation was found between the JH during the SPJ and the maximal horizontal velocity of the center of mass (CoM) (r=0.71, p=0.002), the minimum height of the CoM (r=-0.68, p=0.004), the JH during CMJ (r=0.66, p=0.006) and SJ (r=0.74, p=0.001), the range of movement of right knee flexion-extension (r=0.76, p=0.001) and the angular velocity of left shoulder hyperextension (r=0.72, p=0.002). The asymmetry of the SJ revealed differences in angles, angular velocities of the right and left legs and arms, and a significant difference (p=0.001) between the distances of the left and right foot center to the CoM. Results of our study suggest the importance of optimal approach technique to reach a maximal JH in the volleyball SJ. The SJ movement is influenced by general jumping ability.

Oral magnesium therapy, exercise heart rate, exercise tolerance, and myocardial function in coronary artery disease patients.

BACKGROUND: Previous studies have demonstrated that in patients with coronary artery disease (CAD) upward deflection of the heart rate (HR) performance curve can be observed and that this upward deflection and the degree of the deflection are correlated with a diminished stress dependent left ventricular function. Magnesium supplementation improves endothelial function, exercise tolerance, and exercise induced chest pain in patients with CAD. PURPOSE: We studied the effects of oral magnesium therapy on exercise dependent HR as related to exercise tolerance and resting myocardial function in patients with CAD. METHODS: In a double blind controlled trial, 53 male patients with stable CAD were randomised to either oral magnesium 15 mmol twice daily (n = 28, age 61+/-9 years, height 171+/-7 cm, body weight 79+/-10 kg, previous myocardial infarction, n = 7) or placebo (n = 25, age 58+/-10 years, height 172+/-6 cm, body weight 79+/-10 kg, previous myocardial infarction, n = 6) for 6 months. Maximal oxygen uptake (VO2max), the degree and direction of the deflection of the HR performance curve described as factor k<0 (upward deflection), and the left ventricular ejection fraction (LVEF) were the outcomes measured. RESULTS: Magnesium therapy for 6 months significantly increased intracellular magnesium levels (32.7+/-2.5 v 35.6+/-2.1 mEq/l, p<0.001) compared to placebo (33.1+/-3.1.9 v 33.8+/-2.0 mEq/l, NS), VO2max (28.3+/-6.2 v 30.6+/-7.1 ml/kg/min, p<0.001; 29.3+/-5.4 v 29.6+/-5.2 ml/kg/min, NS), factor k (-0.298+/-0.242 v -0.208+/-0.260, p<0.05; -0.269+/-0.336 v -0.272+/-0.335, NS), and LVEF (58+/-11 v 67+/-10%, p<0.001; 55+/-11 v 54+/-12%, NS). CONCLUSION: The present study supports the intake of oral magnesium and its favourable effects on exercise tolerance and left ventricular function during rest and exercise in stable CAD patients.

Effects of exercise on plasma high-density lipoprotein cholesteryl ester metabolism in male and female miniature swine.

We studied the effects of exercise on high-density lipoprotein (HDL) cholesteryl ester (CE) metabolism in 6 male and 6 female miniature pigs fed a commercial swine diet supplemented with cholesterol and fat. The diets were fed for a total period of 20 weeks. During the last 12 weeks of the feeding period, the pigs were exercised on a motorized treadmill 5 days per week for 45 min/d at a speed of 9.5 to 10.0 km/h at 0% grade. Homologous HDL preparations were radiolabeled with cholesteryl (1-14C)oleate and intravenously administered to the pigs, followed by blood sampling at the appropriate time points and measurement of radiolabeled HDL CE. This was performed while the animals were sedentary and after the exercise period. Plasma cholesterol increased after the exercise protocol from 7.21 +/- 1.90 to 8.50 +/- 2.81 mmol/L (mean +/- SD, n = 6) in the females and from 8.11 +/- 3.61 to 10.07 +/- 3.61 in the males. HDL CE transport rates in female pigs were significantly lower (23%) after the exercise protocol (118 +/- 14 v 91 +/- 14 micromol/h/L plasma). HDL CE transport rates in the males were also lower (11%) after exercise (90 +/- 20 v 80 +/- 18 micromol/h/L plasma), but this effect was not statistically significant. Further, the residence time or life span of HDL CE was significantly longer after the exercise protocol in both male and female pigs. Thus, the results of this study suggest that exercise reduces the transport rate of HDL CE and prolongs the life span of HDL CE in hypercholesterolemic pigs.

Determination of maximal lactate steady state response in selected sports events.

Maximal lactate steady state (MLSS) refers to the upper limit of blood lactate concentration indicating an equilibrium between lactate production and lactate elimination during constant workload. The aim of the present study was to investigate whether different levels of MLSS may explain different blood lactate concentration (BLC) levels at submaximal workload in the sports events of rowing, cycling, and speed skating. Eleven rowers (mean +/- SD, age 20.1 +/- 1.5 yr, height 188.7 +/- 6.2 cm, weight 82.7 +/- 8.0 kg), 16 cyclists and triathletes (age 23.6 +/- 3.0 yr, height 181.4 +/- 5.6 cm, weight 72.5 +/- 6.2 kg), and 6 speed skaters (age 23.3 +/- 6.6 yr, height 179.5 +/- 7.5 cm, weight 73.2 +/- 5.6 kg) performed an incremental load test to determine maximal workload and several submaximal 30-min constant workloads for MLSS measurement on a rowing ergometer, a cycle ergometer, and on a speed-skating track. Maximal workload was higher (P < or = 0.05) in rowing (416.8 +/- 46.2 W) than in cling (358.6 +/- 34.4 W) and speed skating (383.5 +/- 40.9 W). The level of MLSS differed (P < or = 0.001) in rowing (3.1 +/- 0.5 mmol.l-1), cycling (5.4 +/- 1.0 mmol.l-1), and in speed skating (6.6 +/- 0.9 mmol.l-1). MLSS workload was higher (P < or = 0.05) in rowing (316.2 +/- 29.9 W) and speed skating (300.5 +/- 43.8 W) than in cycling (257.8 +/- 34.6 W). No differences (P > 0.05) in MLSS workload were found between speed skating and rowing. MLSS workload intensity as related to maximal workload was independent (P > 0.05) of the sports event: 76.2% +/- 5.7% in rowing, 71.8% +/- 4.1% in cycling, and 78.1% +/- 4.4% in speed skating. Changes in MLSS do not respond with MLSS workload, the MLSS workload intensity, or with the metabolic profile of the sports event. The observed differences in MLSS and MLSS workload may correspond to the sport-specific mass of working muscle.

Intermediate and long-term anaerobic performance of elite Alpine skiers.

Physiological requirements of Alpine skiing, demanding power from both aerobic and anaerobic sources, were first reported in 1965 by Bengt Saltin and coworkers. An update on the physiology of Alpine skiing was presented by Karlsson and colleagues in 1978, and their work remains a benchmark for most current research dealing with Alpine skiers. These works have identified muscular strength and complex motor skill abilities as essential to the competitive ski racer. The energy demands of Alpine ski racing dominate the range between 45 and 2 min. Since the late 1970s, many researchers have reported a variety of tests that associate test scores to skiing performance. Traditionally, short tests of anaerobic power such as the 30-s Wingate test have been used to reflect anaerobic capacity. Only recently have researchers and coaches begun to question whether a test that is shorter in duration than most skiing performances can estimate anaerobic power as it relates to Alpine ski racing. This study reviews current literature relative to physiological requirements for Alpine skiing as well as relating 18 nationally ranked male (N = 10) and female (N = 8) Alpine ski racers' USSA national points lists for slalom and giant slalom to power measures from 30-s and 90-s Wingate cycle ergometer tests. Further directions of physiological research in Alpine skiing are also offered.

Marathons in altitude.

PURPOSE: We examined the effect of altitude up to 5200 m on marathon (42,195 m) performances. METHODS: Eight elite and four good runners participated in a marathon at 4300-m altitude (A1), and five elite runners participated both in A1 and in a marathon at 5200-m altitude (A2). The maximal aerobic power (VO2max) was determined indirectly in altitude during A1 and A2 expeditions from the scores of a 12-min running test. The fractions of VO2max utilized during both races were calculated from the linear relationship between running speed and VO2 described by Costill and Fox (1969). RESULTS: VO2max significantly decreases with altitude (P<0.001). We found a linear relationship (R2 = 0.73, P<0.001) between the speed of each participant in the sea level marathon and the speed of A1. The mean difference between the sea level and the A1 speed was 35+/-9% (P<0.001). In A1, elite runners utilized 63+/-8% whereas good runners utilized 52+/-8% of VO2max (P<0.001). The five elite runners utilized 74+/-6%; 67+/-1% (P< 0.01), and 71+/-3% (P<0.01) of their VO2max at sea level, A1, and A2, respectively. In Al, the mean heart rate (HR) was higher in elite than in good runners (P<0.001), whereas the percentage of maximum theoretical HR was 83+/-3% and 81+/-5%, respectively (P>0.05). CONCLUSIONS: Marathon performance in altitude is mainly affected by the lower VO2max. The better performance of elite marathoners in altitude compared with good runners was related to the higher % of VO2max maintained during every marathon. The differences between the expected and the observed performances at high altitude depend on the uneven running path and on a poorer economy of running that is related to the higher mechanical work of breathing. The fractional utilization of VO2max seems lowered by acute exposure to altitude and slightly increases with acclimatization.

Heart rate performance curve during incremental cycle ergometer exercise in healthy young male subjects.

In 1992 Conconi et al. (20) presented an indirect and noninvasive method for the determination of anaerobic threshold (AnT) in an incremental field test for runners. This noninvasive method for the determination of anaerobic threshold is dependent on the occurrence of a deflection of the heart rate performance curve (HRPC). The aim of our study was to evaluate the degree and direction of the deflection of the HRPC and the relationship of the heart rate threshold (HRT) to the lactate turn point in a group of 227 healthy young subjects (age: 23 +/- 4 yr). The subjects were divided into three groups by means of second degree polynomial fitting (GI: regular deflection, kHR > 0.1; G II: no deflection, 0 < kHR < 0.1; G II: inverse deflection, k < -0.1). No significant differences between the groups were found in the anthropometric data or in the power output and the blood lactate concentration at both the first (LTP1) and second (LTP2) lactate turn points and at maximum performance (Pmax). Using the method of Conconi et al. (20), 85.9% of the subjects showed a "regular" deflection, 6.2% showed no deflection at all, and 7.9% showed even an inverted deflection of the HRPC. An HRT could be obtained in both G I and G III, and power output at HRT was not significantly different in comparison to that at the LTP2. No HRT could be assessed in G II. The heart rate at HRT and the LTP2 were significantly lower in G III compared with G I. The phenomenon of heart rate break point may be attractive in training regulation, but its application is limited because a heart rate deflection cannot be found even in young subjects in some cases.

Monitoring strength training: neuromuscular and hormonal profile.

PURPOSE: This study investigated changes induced by a single heavy resistance training session on neuromuscular and endocrine systems in trained athletes, using the same exercises for training and testing. METHODS: Five different groups volunteered: track and field male sprinters (MS, N = 6), track and field female sprinters (FS, N = 6), body builders (BB, N = 6), and weight lifters performing low-repetition exercise (WLL, N = 4) and high-repetition exercise (WLH, N = 4). In training, the work performed during half and full squat exercise was monitored for mechanical power output as well as EMG analysis on leg extensor muscles of the subjects belonging to the MS, FS, and BB groups. Just before and immediately after the training session, venous blood samples were obtained for RIA determination of testosterone (T), cortisol (C), lutropin (LH), human prolactin (PRL), and follitropin (FSH) in FS and MS. In the other three groups (BB, WLH, and WLL), the hormonal profile was limited to T and human growth hormone (hGH) only. RESULTS: After training the power developed in full squat demonstrated a statistically significant decrease (P < 0.01) in MS and no changes in FS. The EMG activity remained constant during the training session. Consequently, the EMG/Power ratio increased in both MS and FS, although only in MS a statistical significance was noted (P < 0.05). In MS immediately after the session the levels of C, T, and LH were significantly lower (P < 0.05). No changes were found in FS. In both groups and in BB significant negative correlation was found between changes in T level and EMG/Power ratio in half squat performance. CONCLUSIONS: It is likely that adequate T level may compensate the effect of fatigue in FT fibers by ensuring a better neuromuscular efficiency.

Competition performance and basal nocturnal catecholamine excretion in cross-country skiers.

UNLABELLED: Objective techniques to determine whether an athlete is optimally prepared for a competition are virtually nonexistent. Preparedness for sports competition is commonly judged through the experience of the athletes and their coaches. Evidence from overtraining studies suggests that catecholamine (Cat) excretion rates may correlate with performance in athletes. PURPOSE: We therefore attempted to determine whether a relationship existed between performances of world-class cross-country skiers and basal nocturnal Cat excretion (BNCE). METHOD: During the Cross-Country Skiing World Championships, we determined BNCE in nine cross-country skiers of the Swiss national team by measuring free Cat concentration (dopamine = D, norepinephrine = NE, epinephrine = E) in morning urine samples, using high performance liquid chromatography. Expert judgments of competition performance (ECP) were assessed by two experienced professional coaches of the national team by using an 11-step scale. RESULTS: The BNCE correlated significantly with ECP in cross-country skiers (r2 = 0.84 and P < for NE; r2 = 0.86 and P < 0.001 for D). Athletes who had their best absolute competition results (ACR) showed the highest NE and D concentrations. CONCLUSION: These data suggest that competitive cross-country skiers with higher D and NE excretion may reach better competition levels compared with those with lower levels. Measures of BNCE provide objective information about competition performance, which may benefit athletes in their precompetition preparation.

Change in left atrial and ventricular dimensions during and immediately after exercise.

PURPOSE: The aim of this study was to evaluate differences in the left atrial (LAD), total ventricular end-diastolic (TEDD), end-systolic diameters (TESD), and left ventricular shortening fraction (SF) compared with heart rate (HR) and systolic blood pressure (SBP) during exercise and recovery. METHODS: Healthy young male (N = 15) and female (N = 16) subjects performed an incremental cycle ergometer test in upright position, and three phases of energy supply were defined by means of blood lactate concentration (LA) and respiratory gas exchange variables (I: aerobic; II: aerobic-anaerobic transition; III: anaerobic). Subjects were required to rest their arms on a steering bar and to lean their upper body forward; two dimensional (2-D) echocardiograms were obtained over the left parasternal area at rest (R), at the end of each phase, immediately within 15 s post, and 6 min after exercise (6 min). By using VINGMED's "Anatomical M-Mode," it was possible to extract M-Mode Sweeps from stored 2-D-Loops and perform the M-Mode measurement. RESULTS: In contrast to the significant decrease in TEDD and TESD from III to 15 s up to resting values and the significant increase in SF from III to 15 s, the moderate decrease in HR immediately post exercise (15 s) was not significant. The SBP showed a significantly decrease from III to 15 s; in contrast to TEDD, TESD, and SF, the values at 15 s were comparable with the values at II. For LAD, significant increase during exercise and a decrease during recovery were observed. Sex-specific differences of changes in measured variables could not be found. CONCLUSION: We concluded that post exercise measurement of left ventricular and atrial dimensions or SF were not valid to describe heart function at maximal exercise although immediately post exercise HR was near maximal level.

Parasympathetic receptor blockade and the heart rate performance curve.

Parasympathetic receptor blockade and the heart rate performance curve. Med. Sci Sports Sci., Vol. 30. No. 2, pp. 229-233, 1998. The aim of the present study was to investigate the influence of parasympathetic receptor blockade on the heart rate performance curve (HRPC). Twenty healthy male subjects performed a first cycle ergometer test (F), showing a HRPC deflection of varying degree and direction. Subjects then in random order performed two additional cycle ergometer tests, one with atropine (A) and the other with placebo (P). Two lactate turn points (LTP1, and LTP2) were determined by means of linear regression turn point analysis. The degree and direction of the deflection of the HRPC was calculated mathematically as factor kHR (kHR>0 = downsloping of HPRC; kHR<0 = upsloping of HRPC). In comparison with that in F and P, HR in A was significantly higher at rest, LTP1, LTP2, and during recovery, but not at Power(max). An upsloping deflection of the HRPC was seen in only five cases in F and P, whereas in A 10 cases were observed (P < 0.05). In A, kHR was significantly lower than in F and P. A significant correlation for kHR was found among F, P, and A. Independent from parasympathetic receptor blockade and the HR at Power(max), the HR at LTP2 was lower in cases with negative kHR (upsloping). In A as well as in P a significant correlation was observed between kHR and HR at LTP2. The individual time course of HRPC is reproducible and may be independent of parasympathetic activity.

The heart rate turn point reliability and methodological aspects.

PURPOSE: The aim of the study was to test protocol variations on the heart rate performance curve (HRPC) and the heart rate turn point (HRTP) according to Conconi et al. (1996). Respiratory gas exchange variables were used to define three phases of energy supply (I, II, III). METHODS: Eighteen healthy young male subjects performed 4 tests (T1-T4). T1: initial speed of 6 km x h(-1) followed by increments of 0.6 km x h(-1) every 60 s. Subjects were than randomized for the next three tests. T2: initial speed 5.6 km x h(-1) followed by increments of 0.2 km x h(-1) every 20 s; T3: similar to T2, in the second half of phase III acceleration (S) was increased. T4: like T2, at the beginning of phase III, S was increased. No differences were found in the degree of the deflection of the HRPC expressed as factor kHR between T1 (0.228 +/- 0.225) and T2 (0.248 +/- 0.231) but a significant increase was found in T3 (0.533 +/- 0.248) and T4 (0.770 +/- 0.258). RESULTS: The modifications of the protocol (T3 and T4) systematically influenced the deflection of the HRPC, but kHR was highly reproducible in all tests. Eleven subjects showed degrees of deflection in the HRPC in all tests. There were no significant differences for S, HR, and VO2 at the HRTP. An HRTP was not found in seven subjects in neither T1 or T2; however, in T3 and T4, these seven subjects showed a deflection of HRPC resulting from the protocol. The HRTP was found to be dependent on the start of the acceleration in phase III. In cases with a linear time course in the HRPC in T1 and T2, in T3 an HRTP was found at 15.6 km x h(-1) and in T4 at 13.6 km x h(-1) , respectively. CONCLUSION: The Conconi test protocol with an accelerated increase in S in the final phase of the test has a major influence on the occurrence of the HRTP in cases of near linear HRPC.

The heart rate performance curve and left ventricular function during exercise in patients after myocardial infarction.

PURPOSE: The aim of the study was to investigate the heart rate turn point (HRTP) in the time course of the heart rate performance curve (HRPC) in patients after myocardial infarction, and the relationship between the HRTP, the left ventricular function, and the second lactate turn point (LTP2). METHODS: We studied the degree and the direction of the HRPC and the left ventricular ejection fraction (LVEF) in 49 male patients 57 +/- 8 d after their first posterior wall infarction (MI). An incremental cycle ergometer test was performed and three phases of energy supply were defined (I: aerobic; II: aerobic-anaerobic transition; III: anaerobic) via blood lactate LA concentration. HRTP and LVEF-turn points (LVEFTP) were assessed by linear turn point analysis. The degree and direction of the deflection of HRPC were described as factor k (k > 0.1: downward deflection; -0.1 < k < 0.1: linear time curse; k < -0.1: upward deflection). The LVEF was determined by RNA. The difference between Pmax and LTP2 was calculated for LVEF (delta LVEF). RESULTS: An HRTP could be found in 44 and a LVEFTP in 47 cases. The HRTP occurred at 85 +/- 17 Watt (W), which correlated (r = 0.95; P < 0.001) with the LTP2 (84 +/- 17 W) and the LVEFTP (84 +/- 17 W, r = 0.93; P < 0.001). From LTP2 to Pmax a significant decrease in LVEF was found. There was a correlation between the percentage of HRmax at the HRTP and k (r = 0.70), as well as delta LVEF (r = 0.56). CONCLUSIONS: To prevent myocardial overloading, it seems to be useful to determine the HRTP, which indicate the workload where LVEF decreases.

%HRmax target heart rate is dependent on heart rate performance curve deflection.

UNLABELLED: The percent of maximal heart rate (%HRmax) model is widely used to determine training intensities in healthy subjects and patients when prescribing training intensities in these groups of subjects. PURPOSE: The aim of the study was to investigate the influence of the time course of the heart rate performance curve (HRPC) on the accuracy of target training heart rate. METHODS: Sixty-two young healthy male subjects performed an incremental cycle ergometer exercise test until voluntary exhaustion. Subjects were then divided into four groups according to the time course of the HRPC. Groups were classified in regular HR response (kHR2 > 0.2), indifferent HR response (0 < kHR2 < 0.2), linear HR response (kHR2 = 0), and inverted HR response (kHR2 < 0). The first and the second lactate turn point (LTP1, LTP2) as well as the heart rate turn point (HRTP) were determined as submaximal markers of performance. Linear regression lines were calculated for HR in the three regions of energy supply defined by LTP1 and LTP2. RESULTS: HR at LTP1 and HRmax was not significantly different between all four groups. HR at LTP2 was dependent on the time course of the HRPC and was significantly lower (P < 0.05) as kHR2 decreased. Power output and blood lactate concentration at LTP1, LTP2 and maximal workload (Pmax) were not significantly different between the groups. CONCLUSION: From our data, we conclude that target training HR detected by means of the %HRmax method may be overestimated in cases where the HR response is not regular, as it was found in many of our subjects.

A physiological profile of tennis match play.

PURPOSE: The aim of this investigation was to examine physiological demands of single match play in tennis. METHODS: 20 players performed 10 matches of 50 min. Respiratory gas exchange measures (RGEM) and heart rates (HR) were measured using two portable systems. Lactate concentration was determined after each game. The average oxygen uptake (VO2) of 270 games was 29.1 +/- 5.6 mL.kg-1.min-1 (51.1 +/- 10.9% of VO2max). Average VO2 for a game ranged from 10.4 to 47.8 mL.kg-1.min-1 (20.4 and 86.8% of VO2max). Average lactate concentration (LA) was 2.07 +/- 0.9 mmol.L-1 (ranging from 0.7 to 5.2 mmol.L-1). Furthermore, we monitored the duration of rallies (DR), the effective playing time (EPT), and the stroke frequency (SF). The average values of 270 games were DR: 6.4 +/- 4.1 s, EPT: 29.3 +/- 12.1%, SF: 42.6 +/- 9.6 shots.min-1. RESULTS: Multiple regression revealed that the DR was the most promising variable for the determination of VO2 in match play (r = 0.54). The body surface area (BSA) and EPT were also entered into the calculation model. In games of two defensive players, VO2 was significantly higher than in games with at least one offensive player. CONCLUSION: Our results suggest that energy demands of tennis matches are significantly influenced by DR. The highest average VO2 of a game of 47.8 mL.kg-1.min-1 may be regarded as a guide to assess endurance capacity required to sustain high-intensity periods of tennis matches compared with average VO2 of 29.1 mL.kg-1.min-1 for the 270 games. Our results suggest that proper conditioning is advisable especially for players who prefer to play from the baseline.

Left ventricular function in response to the transition from aerobic to anaerobic metabolism.

The purpose of this investigation was to study myocardial function at rest, during three phases of energy supply, and during recovery. Radionuclide angiography was performed during the aerobic phase (phase I, rest-first lactate increase), the aerobic-anaerobic transition phase (phase II, first lactate increase-second lactate increase), the anaerobic phase (phase III, second lactate increase-maximal work performance (Pmax)), and during recovery. Thirty-eight male patients (59 +/- 7 d after myocardial infarction) were compared with 19 healthy control subjects and 21 sport students of comparable age. Left ventricular ejection fraction (LVEF) increased from rest to phase I and from phase I to phase II in sports students and control subjects. During phase III, LVEF did not change significantly in sports students, but it decreased significantly in control subjects. This is in contrast to the patients, who showed an increase of LVEF from resting values (47 +/- 3%) to phase I (50 +/- 1%), no change during phase II (51 +/- 2%), and a decrease to resting values (45 +/- 2) during phase III. All subjects showed an increase in stroke volume (SV) during phase I and II, reaching a maximum at phase II. This was evidenced by an improvement of the systolic function with a constant left ventricular end-diastolic volume (EDV) in control subjects and sports students. In contrast, an improved SV in patients was achieved through an increase in EDV and a less distinct increase in the left ventricular end-systolic volume (ESV). Maximal LVEF values were measured during the first 90 s of recovery in all subjects. Values during recovery are not representative of load dependent myocardial function. This increase in LVEF does not cause an increase in cardiac output but is a consequence of changes in the EDV and ESV, which decrease again immediately after the end of exercise performance.

Off seasonal and pre-seasonal assessment of circulating energy sources during prolonged running at the anaerobic threshold in competitive triathletes.

OBJECTIVES: To compare changes in circulating energy sources during prolonged exercise in off season (OS) and pre-season (PS) training of triathletes. METHODS: Nine athletes of the Swiss national triathlon team (three female, mean (SD) age 28.7 (4.9) years, height 169.8 (6.0) cm, weight 57.0 (6.2) kg, VO(2)max 66.5 (5.3) ml/min/kg; six male, mean (SD) age 24.0 (4.1) years, height 181.4 (6.9) cm, weight 73.5 (6.0) kg, VO(2)max 75.9 (4.9) ml/min/kg) were tested twice (2.5 months apart) during a 25 km aerobic capacity test run at the end of the OS and just before the season. The average training load during the OS was 9.9 h/week, and this increased to 14.4 h/week in the PS. With heart rates as reference, exercise intensity during the aerobic capacity test was 97.0 (4.9)% of the anaerobic threshold and 91.2 (4.5)% of VO(2)max. Blood samples were collected before, during, and after the aerobic capacity test. Samples were collected every 5 km during three minute rest intervals. RESULTS: Blood was analysed for triglyceride (TG), free fatty acids, cholesterol, high density lipoprotein cholesterol, glucose, insulin, lactate, and changes in plasma volume. A two factor (season by distance) repeated measures analysis of variance revealed an increase in capacity for prolonged exercise in the PS by a decrease in running intensity during the aerobic capacity test (% of speed at 2.0 mmol/l lactate threshold, p = 0.008), an increase in running speed at the anaerobic threshold (p = 0.003) and at 4.0 and 2.0 mmol/l (p<0.001) of the lactate threshold. A significant season by distance interaction was found for TG (p<0.001). TG concentrations peaked at 5 km and decreased logarithmically throughout the OS (1.48 (0.34) to 0.86 (0.20) mmol/l) and PS (1.90 (0.31) to 0.73 (0.18) mmol/l) tests. From the OS to the PS, there was an increase in the difference in TG at 5-15 km with a concomitant increase at 2.0 mmol/l of the lactate threshold. The peak TG concentrations at 5 km followed by a logarithmic decrease suggest that TG may also provide circulating energy. A greater logarithmic decrease in TG occurred in the PS than in the OS, indicating a higher rate of use. There was an increase in the difference in TG at 5-15 km similar to the increase in the speed at 2.0 mmol/l of the lactate threshold between the two seasons. Glucose, insulin, lactate, and free fatty acids were similar in the two seasons. CONCLUSION: Free fatty acid and TG concentrations were much higher than expected, and the two training seasons showed significantly different patterns of TG concentration during prolonged running. These responses may be related to aerobic capacity of prolonged exercise.