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Atg16L1 T300A variant decreases selective autophagy resulting in altered cytokine signaling and decreased antibacterial defense.
Lassen, Kara G; Kuballa, Petric; Conway, Kara L; Patel, Khushbu K; Becker, Christine E; Peloquin, Joanna M; Villablanca, Eduardo J; Norman, Jason M; Liu, Ta-Chiang; Heath, Robert J; Becker, Morgan L; Fagbami, Lola; Horn, Heiko; Mercer, Johnathan; Yilmaz, Omer H; Jaffe, Jacob D; Shamji, Alykhan F; Bhan, Atul K; Carr, Steven A; Daly, Mark J; Virgin, Herbert W; Schreiber, Stuart L; Stappenbeck, Thaddeus S; Xavier, Ramnik J.
Afiliación
  • Lassen KG; Broad Institute, Cambridge, MA 02142;Center for Computational and Integrative Biology, Massachusetts General Hospital, Boston, MA 02114;
  • Kuballa P; Broad Institute, Cambridge, MA 02142;Center for Computational and Integrative Biology, Massachusetts General Hospital, Boston, MA 02114;
  • Conway KL; Broad Institute, Cambridge, MA 02142;Center for Computational and Integrative Biology, Massachusetts General Hospital, Boston, MA 02114;Gastrointestinal Unit, Massachusetts General Hospital, Harvard Medical School, Boston, MA 02114;
  • Patel KK; Department of Pathology and Immunology, Washington University School of Medicine, St. Louis, MO 63110;
  • Becker CE; Center for Computational and Integrative Biology, Massachusetts General Hospital, Boston, MA 02114;
  • Peloquin JM; Center for Computational and Integrative Biology, Massachusetts General Hospital, Boston, MA 02114;Gastrointestinal Unit, Massachusetts General Hospital, Harvard Medical School, Boston, MA 02114;
  • Villablanca EJ; Broad Institute, Cambridge, MA 02142;Center for Computational and Integrative Biology, Massachusetts General Hospital, Boston, MA 02114;Gastrointestinal Unit, Massachusetts General Hospital, Harvard Medical School, Boston, MA 02114;
  • Norman JM; Department of Pathology and Immunology, Washington University School of Medicine, St. Louis, MO 63110;
  • Liu TC; Department of Pathology and Immunology, Washington University School of Medicine, St. Louis, MO 63110;
  • Heath RJ; Broad Institute, Cambridge, MA 02142;Center for Computational and Integrative Biology, Massachusetts General Hospital, Boston, MA 02114;
  • Becker ML; Department of Pathology and Immunology, Washington University School of Medicine, St. Louis, MO 63110;
  • Fagbami L; Broad Institute, Cambridge, MA 02142;
  • Horn H; Broad Institute, Cambridge, MA 02142;Department of Surgery, Massachusetts General Hospital, Harvard Medical School, Boston, MA 02114;
  • Mercer J; Broad Institute, Cambridge, MA 02142;
  • Yilmaz OH; Koch Institute for Integrative Cancer Research and Department of Biology, Massachusetts Institute of Technology, Cambridge, MA 02139;Pathology Department, Massachusetts General Hospital, Harvard Medical School, Boston, MA 02114;
  • Jaffe JD; Broad Institute, Cambridge, MA 02142;
  • Shamji AF; Center for the Science of Therapeutics, Broad Institute, Cambridge, MA 02142;
  • Bhan AK; Pathology Department, Massachusetts General Hospital, Harvard Medical School, Boston, MA 02114;Center for the Study of Inflammatory Bowel Disease, Massachusetts General Hospital, Boston, MA 02114;
  • Carr SA; Broad Institute, Cambridge, MA 02142;
  • Daly MJ; Broad Institute, Cambridge, MA 02142;Center for the Study of Inflammatory Bowel Disease, Massachusetts General Hospital, Boston, MA 02114;Analytic and Translational Genetics Unit, Massachusetts General Hospital, Boston, MA 02114;
  • Virgin HW; Department of Pathology and Immunology, Washington University School of Medicine, St. Louis, MO 63110;Department of Molecular Microbiology, Washington University School of Medicine, St. Louis, MO 63110; and.
  • Schreiber SL; Center for the Science of Therapeutics, Broad Institute, Cambridge, MA 02142;Department of Chemistry and Chemical Biology, Harvard University, Cambridge, MA 02138 stuart_schreiber@harvard.edu xavier@molbio.mgh.harvard.edu.
  • Stappenbeck TS; Department of Pathology and Immunology, Washington University School of Medicine, St. Louis, MO 63110;
  • Xavier RJ; Broad Institute, Cambridge, MA 02142;Center for Computational and Integrative Biology, Massachusetts General Hospital, Boston, MA 02114;Gastrointestinal Unit, Massachusetts General Hospital, Harvard Medical School, Boston, MA 02114;Center for the Study of Inflammatory Bowel Disease, Massachusetts Ge
Proc Natl Acad Sci U S A ; 111(21): 7741-6, 2014 May 27.
Article en En | MEDLINE | ID: mdl-24821797
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Texto completo: 1 Colección: 01-internacional Banco de datos: MEDLINE Asunto principal: Infecciones por Salmonella / Proteínas Portadoras / Enfermedad de Crohn / Células de Paneth / Polimorfismo de Nucleótido Simple Tipo de estudio: Prognostic_studies Límite: Animals Idioma: En Revista: Proc Natl Acad Sci U S A Año: 2014 Tipo del documento: Article
Texto completo
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Texto completo: 1 Colección: 01-internacional Banco de datos: MEDLINE Asunto principal: Infecciones por Salmonella / Proteínas Portadoras / Enfermedad de Crohn / Células de Paneth / Polimorfismo de Nucleótido Simple Tipo de estudio: Prognostic_studies Límite: Animals Idioma: En Revista: Proc Natl Acad Sci U S A Año: 2014 Tipo del documento: Article