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Decoupling of the PI3K Pathway via Mutation Necessitates Combinatorial Treatment in HER2+ Breast Cancer.
Korkola, James E; Collisson, Eric A; Heiser, Laura; Oates, Chris; Bayani, Nora; Itani, Sleiman; Esch, Amanda; Thompson, Wallace; Griffith, Obi L; Wang, Nicholas J; Kuo, Wen-Lin; Cooper, Brian; Billig, Jessica; Ziyad, Safiyyah; Hung, Jenny L; Jakkula, Lakshmi; Feiler, Heidi; Lu, Yiling; Mills, Gordon B; Spellman, Paul T; Tomlin, Claire; Mukherjee, Sach; Gray, Joe W.
Afiliación
  • Korkola JE; Oregon Health Sciences University, Department of Biomedical Engineering, Portland, Oregon, United States of America.
  • Collisson EA; University of California San Francisco, Division of Heme/Onc, San Francisco, California, United States of America.
  • Heiser M; Oregon Health Sciences University, Department of Biomedical Engineering, Portland, Oregon, United States of America.
  • Oates C; Netherlands Cancer Institute, Division of Biochemistry, Amsterdam, The Netherlands; University of Warwick, Centre for Complexity Science, Coventry, United Kingdom.
  • Bayani N; Lawrence Berkeley National Laboratories, Life Sciences Division, Berkeley, California, United States of America.
  • Itani S; University of California, Berkeley, Department of Electrical Engineering and Computer Sciences, Berkeley, California, United States of America.
  • Esch A; Oregon Health Sciences University, Department of Biomedical Engineering, Portland, Oregon, United States of America.
  • Thompson W; Oregon Health Sciences University, Department of Biomedical Engineering, Portland, Oregon, United States of America.
  • Griffith OL; Lawrence Berkeley National Laboratories, Life Sciences Division, Berkeley, California, United States of America.
  • Wang NJ; Oregon Health Sciences University, Department of Biomedical Engineering, Portland, Oregon, United States of America.
  • Kuo WL; Lawrence Berkeley National Laboratories, Life Sciences Division, Berkeley, California, United States of America.
  • Cooper B; Lawrence Berkeley National Laboratories, Life Sciences Division, Berkeley, California, United States of America.
  • Billig J; Lawrence Berkeley National Laboratories, Life Sciences Division, Berkeley, California, United States of America.
  • Ziyad S; Lawrence Berkeley National Laboratories, Life Sciences Division, Berkeley, California, United States of America.
  • Hung JL; Lawrence Berkeley National Laboratories, Life Sciences Division, Berkeley, California, United States of America.
  • Jakkula L; Lawrence Berkeley National Laboratories, Life Sciences Division, Berkeley, California, United States of America.
  • Feiler H; Oregon Health Sciences University, Department of Biomedical Engineering, Portland, Oregon, United States of America; Lawrence Berkeley National Laboratories, Life Sciences Division, Berkeley, California, United States of America.
  • Lu Y; MD Anderson Cancer Center, Department of Systems Biology, Houston, Texas, United States of America.
  • Mills GB; MD Anderson Cancer Center, Department of Systems Biology, Houston, Texas, United States of America.
  • Spellman PT; Oregon Health Sciences University, Department of Biomedical Engineering, Portland, Oregon, United States of America.
  • Tomlin C; University of California, Berkeley, Department of Electrical Engineering and Computer Sciences, Berkeley, California, United States of America.
  • Mukherjee S; Netherlands Cancer Institute, Division of Biochemistry, Amsterdam, The Netherlands.
  • Gray JW; Oregon Health Sciences University, Department of Biomedical Engineering, Portland, Oregon, United States of America.
PLoS One ; 10(7): e0133219, 2015.
Article en En | MEDLINE | ID: mdl-26181325
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Texto completo: 1 Colección: 01-internacional Banco de datos: MEDLINE Asunto principal: Regulación Neoplásica de la Expresión Génica / Receptor ErbB-2 / Fosfatidilinositol 3-Quinasas / Células Epiteliales / Proteínas Proto-Oncogénicas c-akt / Antineoplásicos Límite: Female / Humans Idioma: En Revista: PLoS One Asunto de la revista: CIENCIA / MEDICINA Año: 2015 Tipo del documento: Article País de afiliación: Estados Unidos
Texto completo
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Texto completo: 1 Colección: 01-internacional Banco de datos: MEDLINE Asunto principal: Regulación Neoplásica de la Expresión Génica / Receptor ErbB-2 / Fosfatidilinositol 3-Quinasas / Células Epiteliales / Proteínas Proto-Oncogénicas c-akt / Antineoplásicos Límite: Female / Humans Idioma: En Revista: PLoS One Asunto de la revista: CIENCIA / MEDICINA Año: 2015 Tipo del documento: Article País de afiliación: Estados Unidos