TLR and RLR Signaling Are Reprogrammed in Opposite Directions after Detection of Viral Infection.

Hotz, Christian; Roetzer, Laurin C; Huber, Thomas; Sailer, Andreas; Oberson, Anne; Treinies, Marina; Heidegger, Simon; Herbst, Tina; Endres, Stefan; Bourquin, Carole

Hotz, Christian; Roetzer, Laurin C; Huber, Thomas; Sailer, Andreas; Oberson, Anne; Treinies, Marina; Heidegger, Simon; Herbst, Tina; Endres, Stefan; Bourquin, Carole.

Afiliación

Hotz C; Department of Medicine, Faculty of Science, University of Fribourg, 1700 Fribourg, Switzerland; Division of Clinical Pharmacology, Center for Integrated Protein Science Munich, Ludwig-Maximilian-University Munich, 80336 Munich, Germany; and.
Roetzer LC; Division of Clinical Pharmacology, Center for Integrated Protein Science Munich, Ludwig-Maximilian-University Munich, 80336 Munich, Germany; and.
Huber T; Division of Clinical Pharmacology, Center for Integrated Protein Science Munich, Ludwig-Maximilian-University Munich, 80336 Munich, Germany; and.
Sailer A; Division of Clinical Pharmacology, Center for Integrated Protein Science Munich, Ludwig-Maximilian-University Munich, 80336 Munich, Germany; and.
Oberson A; Department of Medicine, Faculty of Science, University of Fribourg, 1700 Fribourg, Switzerland;
Treinies M; Department of Medicine, Faculty of Science, University of Fribourg, 1700 Fribourg, Switzerland;
Heidegger S; Division of Clinical Pharmacology, Center for Integrated Protein Science Munich, Ludwig-Maximilian-University Munich, 80336 Munich, Germany; and III Medical Department, Technical University Munich, 81675 Munich, Germany.
Herbst T; Department of Medicine, Faculty of Science, University of Fribourg, 1700 Fribourg, Switzerland;
Endres S; Division of Clinical Pharmacology, Center for Integrated Protein Science Munich, Ludwig-Maximilian-University Munich, 80336 Munich, Germany; and.
Bourquin C; Department of Medicine, Faculty of Science, University of Fribourg, 1700 Fribourg, Switzerland; Division of Clinical Pharmacology, Center for Integrated Protein Science Munich, Ludwig-Maximilian-University Munich, 80336 Munich, Germany; and carole.bourquin@unifr.ch.

J Immunol ; 195(9): 4387-95, 2015 Nov 01.

Article en En | MEDLINE | ID: mdl-26392465

Asunto(s)

ARN Helicasas DEAD-box/inmunología; Transducción de Señal/inmunología; Receptores Toll-Like/inmunología; Virosis/inmunología; Animales; Línea Celular; Línea Celular Tumoral; Proteína 58 DEAD Box; ARN Helicasas DEAD-box/genética; ARN Helicasas DEAD-box/metabolismo; Expresión Génica/efectos de los fármacos; Expresión Génica/inmunología; Immunoblotting; Interferón-alfa/inmunología; Interferón-alfa/metabolismo; Interferón beta/inmunología; Interferón beta/metabolismo; Interleucina-10/inmunología; Interleucina-10/metabolismo; Interleucina-6/inmunología; Interleucina-6/metabolismo; Macrófagos/efectos de los fármacos; Macrófagos/inmunología; Macrófagos/virología; Ratones de la Cepa 129; Ratones Endogámicos C57BL; Ratones Noqueados; Poli I-C/inmunología; Poli I-C/farmacología; Receptores de Reconocimiento de Patrones/inmunología; Reacción en Cadena de la Polimerasa de Transcriptasa Inversa; Virus Sendai/inmunología; Virus Sendai/fisiología; Transducción de Señal/efectos de los fármacos; Receptores Toll-Like/genética; Receptores Toll-Like/metabolismo

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Texto completo: 1 Colección: 01-internacional Banco de datos: MEDLINE Asunto principal: Virosis / Transducción de Señal / Receptores Toll-Like / ARN Helicasas DEAD-box Tipo de estudio: Diagnostic_studies Idioma: En Revista: J Immunol Año: 2015 Tipo del documento: Article

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