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ROS-mediated glucose metabolic reprogram induces insulin resistance in type 2 diabetes.
Dong, Kelei; Ni, Hua; Wu, Meiling; Tang, Ziqing; Halim, Michael; Shi, Dongyun.
Afiliación
  • Dong K; Department of Biochemistry and Molecular Biology, Shanghai Medical College of Fudan University, Free Radical Regulation and Application Research Center of Fudan University, Shanghai 200032, People's Republic of China.
  • Ni H; Department of Biochemistry and Molecular Biology, Shanghai Medical College of Fudan University, Free Radical Regulation and Application Research Center of Fudan University, Shanghai 200032, People's Republic of China.
  • Wu M; Department of Biochemistry and Molecular Biology, Shanghai Medical College of Fudan University, Free Radical Regulation and Application Research Center of Fudan University, Shanghai 200032, People's Republic of China.
  • Tang Z; Department of Biochemistry and Molecular Biology, Shanghai Medical College of Fudan University, Free Radical Regulation and Application Research Center of Fudan University, Shanghai 200032, People's Republic of China.
  • Halim M; Department of Biochemistry and Molecular Biology, Shanghai Medical College of Fudan University, Free Radical Regulation and Application Research Center of Fudan University, Shanghai 200032, People's Republic of China.
  • Shi D; Department of Biochemistry and Molecular Biology, Shanghai Medical College of Fudan University, Free Radical Regulation and Application Research Center of Fudan University, Shanghai 200032, People's Republic of China. Electronic address: dyshi@fudan.edu.cn.
Biochem Biophys Res Commun ; 476(4): 204-211, 2016 08 05.
Article en En | MEDLINE | ID: mdl-27207834

Texto completo: 1 Colección: 01-internacional Banco de datos: MEDLINE Asunto principal: Resistencia a la Insulina / Especies Reactivas de Oxígeno / Diabetes Mellitus Tipo 2 / Glucosa Límite: Animals Idioma: En Revista: Biochem Biophys Res Commun Año: 2016 Tipo del documento: Article

Texto completo: 1 Colección: 01-internacional Banco de datos: MEDLINE Asunto principal: Resistencia a la Insulina / Especies Reactivas de Oxígeno / Diabetes Mellitus Tipo 2 / Glucosa Límite: Animals Idioma: En Revista: Biochem Biophys Res Commun Año: 2016 Tipo del documento: Article