p62-mediated Selective autophagy endows virus-transformed cells with insusceptibility to DNA damage under oxidative stress.

Wang, Ling; Howell, Mary E A; Sparks-Wallace, Ayrianna; Hawkins, Caroline; Nicksic, Camri A; Kohne, Carissa; Hall, Kenton H; Moorman, Jonathan P; Yao, Zhi Q; Ning, Shunbin

Wang, Ling; Howell, Mary E A; Sparks-Wallace, Ayrianna; Hawkins, Caroline; Nicksic, Camri A; Kohne, Carissa; Hall, Kenton H; Moorman, Jonathan P; Yao, Zhi Q; Ning, Shunbin.

Afiliación

Wang L; Department of Internal Medicine, Quillen College of Medicine, East Tennessee State University, Johnson City, TN, United States of America.
Howell MEA; Center of Excellence for Inflammation, Infectious Diseases and Immunity, Quillen College of Medicine, East Tennessee State University, Johnson City, TN, United States of America.
Sparks-Wallace A; Department of Internal Medicine, Quillen College of Medicine, East Tennessee State University, Johnson City, TN, United States of America.
Hawkins C; Department of Internal Medicine, Quillen College of Medicine, East Tennessee State University, Johnson City, TN, United States of America.
Nicksic CA; Department of Internal Medicine, Quillen College of Medicine, East Tennessee State University, Johnson City, TN, United States of America.
Kohne C; Department of Internal Medicine, Quillen College of Medicine, East Tennessee State University, Johnson City, TN, United States of America.
Hall KH; Department of Internal Medicine, Quillen College of Medicine, East Tennessee State University, Johnson City, TN, United States of America.
Moorman JP; Department of Internal Medicine, Quillen College of Medicine, East Tennessee State University, Johnson City, TN, United States of America.
Yao ZQ; Department of Internal Medicine, Quillen College of Medicine, East Tennessee State University, Johnson City, TN, United States of America.
Ning S; Center of Excellence for Inflammation, Infectious Diseases and Immunity, Quillen College of Medicine, East Tennessee State University, Johnson City, TN, United States of America.

PLoS Pathog ; 15(4): e1007541, 2019 04.

Article en En | MEDLINE | ID: mdl-31017975

Asunto(s)

Autofagia; Transformación Celular Viral; Daño del ADN; Infecciones por Virus de Epstein-Barr/patología; Estrés Oxidativo; Proteínas de Unión al ARN/metabolismo; Especies Reactivas de Oxígeno/metabolismo; Linfoma de Burkitt/genética; Linfoma de Burkitt/metabolismo; Linfoma de Burkitt/patología; Linfoma de Burkitt/virología; Cromatina; Reparación del ADN; Infecciones por Virus de Epstein-Barr/genética; Infecciones por Virus de Epstein-Barr/metabolismo; Infecciones por Virus de Epstein-Barr/virología; Herpesvirus Humano 4/fisiología; Humanos; Proteína 1 Asociada A ECH Tipo Kelch/genética; Proteína 1 Asociada A ECH Tipo Kelch/metabolismo; Factor 2 Relacionado con NF-E2/genética; Factor 2 Relacionado con NF-E2/metabolismo; Complejo de la Endopetidasa Proteasomal; Proteínas de Unión al ARN/genética; Transducción de Señal; Ubiquitina/metabolismo; Latencia del Virus

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Texto completo: 1 Colección: 01-internacional Banco de datos: MEDLINE Asunto principal: Autofagia / Daño del ADN / Transformación Celular Viral / Proteínas de Unión al ARN / Especies Reactivas de Oxígeno / Estrés Oxidativo / Infecciones por Virus de Epstein-Barr Límite: Humans Idioma: En Revista: PLoS Pathog Año: 2019 Tipo del documento: Article País de afiliación: Estados Unidos

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