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In Colorectal Cancer Cells With Mutant KRAS, SLC25A22-Mediated Glutaminolysis Reduces DNA Demethylation to Increase WNT Signaling, Stemness, and Drug Resistance.
Wong, Chi Chun; Xu, Jiaying; Bian, Xiqing; Wu, Jian-Lin; Kang, Wei; Qian, Yun; Li, Weilin; Chen, Huarong; Gou, Hongyan; Liu, Dabin; Yat Luk, Simson Tsz; Zhou, Qiming; Ji, Fenfen; Chan, Lam-Shing; Shirasawa, Senji; Sung, Joseph Jy; Yu, Jun.
Afiliación
  • Wong CC; Institute of Digestive Disease and Department of Medicine and Therapeutics, State Key Laboratory of Digestive Disease, Li Ka Shing Institute of Health Sciences, The Chinese University of Hong Kong, Shenzhen Research Institute, Chinese University of Hong Kong, Hong Kong, China. Electronic address: ch
  • Xu J; Institute of Digestive Disease and Department of Medicine and Therapeutics, State Key Laboratory of Digestive Disease, Li Ka Shing Institute of Health Sciences, The Chinese University of Hong Kong, Shenzhen Research Institute, Chinese University of Hong Kong, Hong Kong, China.
  • Bian X; State Key Laboratory for Quality Research in Chinese Medicines, Macau University of Science and Technology, Macao, China.
  • Wu JL; State Key Laboratory for Quality Research in Chinese Medicines, Macau University of Science and Technology, Macao, China.
  • Kang W; Department of Anatomical and Chemical Pathology, Chinese University of Hong Kong, Hong Kong, China.
  • Qian Y; Department of Gastroenterology and Hepatology, Shenzhen University General Hospital, Shenzhen, China.
  • Li W; Institute of Digestive Disease and Department of Medicine and Therapeutics, State Key Laboratory of Digestive Disease, Li Ka Shing Institute of Health Sciences, The Chinese University of Hong Kong, Shenzhen Research Institute, Chinese University of Hong Kong, Hong Kong, China; Department of Surgery,
  • Chen H; Institute of Digestive Disease and Department of Medicine and Therapeutics, State Key Laboratory of Digestive Disease, Li Ka Shing Institute of Health Sciences, The Chinese University of Hong Kong, Shenzhen Research Institute, Chinese University of Hong Kong, Hong Kong, China.
  • Gou H; Institute of Digestive Disease and Department of Medicine and Therapeutics, State Key Laboratory of Digestive Disease, Li Ka Shing Institute of Health Sciences, The Chinese University of Hong Kong, Shenzhen Research Institute, Chinese University of Hong Kong, Hong Kong, China.
  • Liu D; Institute of Digestive Disease and Department of Medicine and Therapeutics, State Key Laboratory of Digestive Disease, Li Ka Shing Institute of Health Sciences, The Chinese University of Hong Kong, Shenzhen Research Institute, Chinese University of Hong Kong, Hong Kong, China.
  • Yat Luk ST; Institute of Digestive Disease and Department of Medicine and Therapeutics, State Key Laboratory of Digestive Disease, Li Ka Shing Institute of Health Sciences, The Chinese University of Hong Kong, Shenzhen Research Institute, Chinese University of Hong Kong, Hong Kong, China.
  • Zhou Q; Institute of Digestive Disease and Department of Medicine and Therapeutics, State Key Laboratory of Digestive Disease, Li Ka Shing Institute of Health Sciences, The Chinese University of Hong Kong, Shenzhen Research Institute, Chinese University of Hong Kong, Hong Kong, China.
  • Ji F; Institute of Digestive Disease and Department of Medicine and Therapeutics, State Key Laboratory of Digestive Disease, Li Ka Shing Institute of Health Sciences, The Chinese University of Hong Kong, Shenzhen Research Institute, Chinese University of Hong Kong, Hong Kong, China.
  • Chan LS; Institute of Digestive Disease and Department of Medicine and Therapeutics, State Key Laboratory of Digestive Disease, Li Ka Shing Institute of Health Sciences, The Chinese University of Hong Kong, Shenzhen Research Institute, Chinese University of Hong Kong, Hong Kong, China.
  • Shirasawa S; Department of Cell Biology, Faculty of Medicine, Fukuoka University, Fukuoka, Japan.
  • Sung JJ; Institute of Digestive Disease and Department of Medicine and Therapeutics, State Key Laboratory of Digestive Disease, Li Ka Shing Institute of Health Sciences, The Chinese University of Hong Kong, Shenzhen Research Institute, Chinese University of Hong Kong, Hong Kong, China.
  • Yu J; Institute of Digestive Disease and Department of Medicine and Therapeutics, State Key Laboratory of Digestive Disease, Li Ka Shing Institute of Health Sciences, The Chinese University of Hong Kong, Shenzhen Research Institute, Chinese University of Hong Kong, Hong Kong, China. Electronic address: ju
Gastroenterology ; 159(6): 2163-2180.e6, 2020 12.
Article en En | MEDLINE | ID: mdl-32814111
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Texto completo: 1 Colección: 01-internacional Banco de datos: MEDLINE Asunto principal: Neoplasias Colorrectales / Colon / Proteínas de Transporte de Membrana Mitocondrial / Mucosa Intestinal Tipo de estudio: Observational_studies / Prognostic_studies / Risk_factors_studies Límite: Animals / Female / Humans / Male País/Región como asunto: Asia Idioma: En Revista: Gastroenterology Año: 2020 Tipo del documento: Article
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Texto completo: 1 Colección: 01-internacional Banco de datos: MEDLINE Asunto principal: Neoplasias Colorrectales / Colon / Proteínas de Transporte de Membrana Mitocondrial / Mucosa Intestinal Tipo de estudio: Observational_studies / Prognostic_studies / Risk_factors_studies Límite: Animals / Female / Humans / Male País/Región como asunto: Asia Idioma: En Revista: Gastroenterology Año: 2020 Tipo del documento: Article