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Age-induced mitochondrial DNA point mutations are inadequate to alter metabolic homeostasis in response to nutrient challenge.
Moore, Timothy M; Zhou, Zhenqi; Strumwasser, Alexander R; Cohn, Whitaker; Lin, Amanda J; Cory, Kevin; Whitney, Kate; Ho, Theodore; Ho, Timothy; Lee, Joseph L; Rucker, Daniel H; Hoang, Austin N; Widjaja, Kevin; Abrishami, Aaron D; Charugundla, Sarada; Stiles, Linsey; Whitelegge, Julian P; Turcotte, Lorraine P; Wanagat, Jonathan; Hevener, Andrea L.
Afiliación
  • Moore TM; Department of Biological Sciences, Dana & David Dornsife College of Letters, Arts, and Sciences, University of Southern California, Los Angeles, CA, USA.
  • Zhou Z; Division of Endocrinology, Diabetes, and Hypertension, Department of Medicine, David Geffen School of Medicine, University of California, Los Angeles, CA, USA.
  • Strumwasser AR; Division of Endocrinology, Diabetes, and Hypertension, Department of Medicine, David Geffen School of Medicine, University of California, Los Angeles, CA, USA.
  • Cohn W; Division of Endocrinology, Diabetes, and Hypertension, Department of Medicine, David Geffen School of Medicine, University of California, Los Angeles, CA, USA.
  • Lin AJ; Department of Psychiatry and Biobehavioral Sciences & The Semel Institute for Neuroscience and Human Behavior, University of California, Los Angeles, CA, USA.
  • Cory K; Division of Endocrinology, Diabetes, and Hypertension, Department of Medicine, David Geffen School of Medicine, University of California, Los Angeles, CA, USA.
  • Whitney K; Division of Endocrinology, Diabetes, and Hypertension, Department of Medicine, David Geffen School of Medicine, University of California, Los Angeles, CA, USA.
  • Ho T; Division of Endocrinology, Diabetes, and Hypertension, Department of Medicine, David Geffen School of Medicine, University of California, Los Angeles, CA, USA.
  • Ho T; Division of Endocrinology, Diabetes, and Hypertension, Department of Medicine, David Geffen School of Medicine, University of California, Los Angeles, CA, USA.
  • Lee JL; Division of Endocrinology, Diabetes, and Hypertension, Department of Medicine, David Geffen School of Medicine, University of California, Los Angeles, CA, USA.
  • Rucker DH; Division of Endocrinology, Diabetes, and Hypertension, Department of Medicine, David Geffen School of Medicine, University of California, Los Angeles, CA, USA.
  • Hoang AN; Division of Endocrinology, Diabetes, and Hypertension, Department of Medicine, David Geffen School of Medicine, University of California, Los Angeles, CA, USA.
  • Widjaja K; Division of Endocrinology, Diabetes, and Hypertension, Department of Medicine, David Geffen School of Medicine, University of California, Los Angeles, CA, USA.
  • Abrishami AD; Division of Endocrinology, Diabetes, and Hypertension, Department of Medicine, David Geffen School of Medicine, University of California, Los Angeles, CA, USA.
  • Charugundla S; Division of Cardiology, Department of Medicine, David Geffen School of Medicine, University of California, Los Angeles, CA, USA.
  • Stiles L; Division of Cardiology, Department of Medicine, David Geffen School of Medicine, University of California, Los Angeles, CA, USA.
  • Whitelegge JP; Division of Endocrinology, Diabetes, and Hypertension, Department of Medicine, David Geffen School of Medicine, University of California, Los Angeles, CA, USA.
  • Turcotte LP; Department of Psychiatry and Biobehavioral Sciences & The Semel Institute for Neuroscience and Human Behavior, University of California, Los Angeles, CA, USA.
  • Wanagat J; Department of Biological Sciences, Dana & David Dornsife College of Letters, Arts, and Sciences, University of Southern California, Los Angeles, CA, USA.
  • Hevener AL; Division of Endocrinology, Diabetes, and Hypertension, Department of Medicine, David Geffen School of Medicine, University of California, Los Angeles, CA, USA.
Aging Cell ; 19(11): e13166, 2020 11.
Article en En | MEDLINE | ID: mdl-33049094
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Texto completo: 1 Colección: 01-internacional Banco de datos: MEDLINE Asunto principal: ADN Mitocondrial / Mitocondrias Hepáticas / Mutación Puntual / Mitocondrias Musculares Tipo de estudio: Prognostic_studies Límite: Animals Idioma: En Revista: Aging Cell Año: 2020 Tipo del documento: Article País de afiliación: Estados Unidos
Texto completo
Imprimir
XML
PubMed Links
Buscar en Google

Texto completo: 1 Colección: 01-internacional Banco de datos: MEDLINE Asunto principal: ADN Mitocondrial / Mitocondrias Hepáticas / Mutación Puntual / Mitocondrias Musculares Tipo de estudio: Prognostic_studies Límite: Animals Idioma: En Revista: Aging Cell Año: 2020 Tipo del documento: Article País de afiliación: Estados Unidos