HMGB1 could restrict 1,3-ß-glucan induced mice lung inflammation by affecting Beclin1 and Bcl2 interaction and promoting the autophagy of epithelial cells.

Zeng, Xinning; Liu, Fangwei; Liu, Kaiyue; Xin, Jiaxuan; Chen, Jie

Zeng, Xinning; Liu, Fangwei; Liu, Kaiyue; Xin, Jiaxuan; Chen, Jie.

Afiliación

Zeng X; Division of Pneumoconiosis, School of Public Health, China Medical University, Shenyang, PR China.
Liu F; Division of Pneumoconiosis, School of Public Health, China Medical University, Shenyang, PR China.
Liu K; Division of Pneumoconiosis, School of Public Health, China Medical University, Shenyang, PR China.
Xin J; Division of Pneumoconiosis, School of Public Health, China Medical University, Shenyang, PR China.
Chen J; Division of Pneumoconiosis, School of Public Health, China Medical University, Shenyang, PR China. Electronic address: jchen@cmu.edu.cn.

Ecotoxicol Environ Saf ; 222: 112460, 2021 Oct 01.

Article en En | MEDLINE | ID: mdl-34243113

Asunto(s)

Proteína HMGB1; Neumonía; Animales; Autofagia; Beclina-1/genética; Células Epiteliales; Glucanos; Proteína HMGB1/genética; Ratones

Palabras clave

1,3-ß-glucan; Autophagy; Beclin1; Epithelial cells; Inflammation

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Texto completo: 1 Colección: 01-internacional Banco de datos: MEDLINE Asunto principal: Neumonía / Proteína HMGB1 Tipo de estudio: Prognostic_studies Límite: Animals Idioma: En Revista: Ecotoxicol Environ Saf Año: 2021 Tipo del documento: Article

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