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Full-length dystrophin deficiency leads to contractile and calcium transient defects in human engineered heart tissues.
Bremner, Samantha B; Mandrycky, Christian J; Leonard, Andrea; Padgett, Ruby M; Levinson, Alan R; Rehn, Ethan S; Pioner, J Manuel; Sniadecki, Nathan J; Mack, David L.
Afiliación
  • Bremner SB; Department of Bioengineering, University of Washington, Seattle, WA, USA.
  • Mandrycky CJ; Institute for Stem Cell and Regenerative Medicine, University of Washington, Seattle, WA, USA.
  • Leonard A; Department of Bioengineering, University of Washington, Seattle, WA, USA.
  • Padgett RM; Institute for Stem Cell and Regenerative Medicine, University of Washington, Seattle, WA, USA.
  • Levinson AR; Institute for Stem Cell and Regenerative Medicine, University of Washington, Seattle, WA, USA.
  • Rehn ES; Department of Mechanical Engineering, University of Washington, Seattle, WA, USA.
  • Pioner JM; Center for Cardiovascular Biology, University of Washington, Seattle, WA, USA.
  • Sniadecki NJ; Institute for Stem Cell and Regenerative Medicine, University of Washington, Seattle, WA, USA.
  • Mack DL; Department of Mechanical Engineering, University of Washington, Seattle, WA, USA.
J Tissue Eng ; 13: 20417314221119628, 2022.
Article en En | MEDLINE | ID: mdl-36003954
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Texto completo: 1 Colección: 01-internacional Banco de datos: MEDLINE Tipo de estudio: Prognostic_studies Idioma: En Revista: J Tissue Eng Año: 2022 Tipo del documento: Article País de afiliación: Estados Unidos
Texto completo
Imprimir
XML
PubMed Links
Buscar en Google

Texto completo: 1 Colección: 01-internacional Banco de datos: MEDLINE Tipo de estudio: Prognostic_studies Idioma: En Revista: J Tissue Eng Año: 2022 Tipo del documento: Article País de afiliación: Estados Unidos