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GNASR201C Induces Pancreatic Cystic Neoplasms in Mice That Express Activated KRAS by Inhibiting YAP1 Signaling.
Ideno, Noboru; Yamaguchi, Hiroshi; Ghosh, Bidyut; Gupta, Sonal; Okumura, Takashi; Steffen, Dana J; Fisher, Catherine G; Wood, Laura D; Singhi, Aatur D; Nakamura, Masafumi; Gutkind, J Silvio; Maitra, Anirban.
Afiliação
  • Ideno N; Department of Translational Molecular Pathology, Sheikh Ahmed Center for Pancreatic Cancer Center, Houston, Texas. Electronic address: NIdeno@mdanderson.org.
  • Yamaguchi H; Department of Translational Molecular Pathology, Sheikh Ahmed Center for Pancreatic Cancer Center, Houston, Texas.
  • Ghosh B; Department of Translational Molecular Pathology, Sheikh Ahmed Center for Pancreatic Cancer Center, Houston, Texas.
  • Gupta S; Department of Translational Molecular Pathology, Sheikh Ahmed Center for Pancreatic Cancer Center, Houston, Texas.
  • Okumura T; Department of Translational Molecular Pathology, Sheikh Ahmed Center for Pancreatic Cancer Center, Houston, Texas.
  • Steffen DJ; Department of Pharmacology, University of California San Diego, La Jolla, California.
  • Fisher CG; Department of Pathology, Sol Goldman Pancreatic Cancer Research Center, Baltimore, Maryland.
  • Wood LD; Department of Pathology, Sol Goldman Pancreatic Cancer Research Center, Baltimore, Maryland; Department of Oncology, Sol Goldman Pancreatic Cancer Research Center, Baltimore, Maryland.
  • Singhi AD; Department of Anatomic Pathology, University of Pittsburgh, Pittsburgh, Pennsylvania.
  • Nakamura M; Department of Surgery and Oncology, Graduate School of Medical Sciences, Kyushu University, Fukuoka, Japan.
  • Gutkind JS; Department of Pharmacology, University of California San Diego, La Jolla, California.
  • Maitra A; Department of Translational Molecular Pathology, Sheikh Ahmed Center for Pancreatic Cancer Center, Houston, Texas.
Gastroenterology ; 155(5): 1593-1607.e12, 2018 11.
Article em En | MEDLINE | ID: mdl-30142336

Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Neoplasias Pancreáticas / Fosfoproteínas / Transdução de Sinais / Proteínas Proto-Oncogênicas p21(ras) / Cromograninas / Neoplasias Císticas, Mucinosas e Serosas / Subunidades alfa Gs de Proteínas de Ligação ao GTP / Carcinoma Ductal Pancreático / Proteínas Adaptadoras de Transdução de Sinal / Mutação Limite: Animals / Humans Idioma: En Revista: Gastroenterology Ano de publicação: 2018 Tipo de documento: Article

Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Neoplasias Pancreáticas / Fosfoproteínas / Transdução de Sinais / Proteínas Proto-Oncogênicas p21(ras) / Cromograninas / Neoplasias Císticas, Mucinosas e Serosas / Subunidades alfa Gs de Proteínas de Ligação ao GTP / Carcinoma Ductal Pancreático / Proteínas Adaptadoras de Transdução de Sinal / Mutação Limite: Animals / Humans Idioma: En Revista: Gastroenterology Ano de publicação: 2018 Tipo de documento: Article